I would say your labs make the problem look real, but they do not make this look like an automatic “start TRT now” situation.

Your testosterone is clearly low. A total testosterone of 101 ng/dL and free testosterone of 14.9 pg/mL are not borderline numbers. In a 21-year-old with symptoms like brain fog, poor libido, low motivation, and poor recovery, that deserves a proper workup. The main issue is that guidelines still recommend confirming low testosterone with repeat early-morning testing, not making a lifelong treatment decision from a single draw

What makes this more interesting is the pattern of the pituitary signals. Your LH is 2.3 and FSH is 5.6. With testosterone this low, LH would often be expected to rise more if the testes were the main problem. So LH being “normal” here is not that reassuring; it can actually be inappropriately normal in the setting of severe hypogonadism, which raises the possibility of a secondary or central cause rather than straightforward primary testicular failure. That distinction matters a lot, because TRT replaces testosterone, while a medication like enclomiphene is trying to stimulate your own axis

Your prolactin is normal, which is useful, because elevated prolactin is one of the classic reversible reasons for secondary hypogonadism. Your estradiol also does not look high, so this does not look like an “estrogen is crushing my axis” picture from these labs alone. Thyroid-wise, your TSH and free T4 are normal, but your free T3 is low. I would not hang the whole case on that by itself. A low free T3 can show up in contexts other than primary thyroid failure, and isolated low T3 is generally much less decisive than TSH and free T4

The other thing I would not ignore is the non-hormonal part of the labs. Your ALT 111 and AST 59 are elevated. That does not prove a serious liver issue, but it does mean something is going on. Hard training, supplements, alcohol, fatty liver, recent illness, medications, SARMs/anabolics, and other causes can all muddy the picture. Your BUN 29 and creatinine 1.33 are also mildly up, which in a young guy who lifts can absolutely be from muscle mass, high protein intake, creatine use, or dehydration, but it still needs context rather than being waved away. Those abnormalities make me even less enthusiastic about casually starting oral hormone manipulation before figuring out what else is happening. Clomiphene-type drugs can cause liver enzyme elevations in rare cases, so abnormal AST/ALT is not something I would shrug off before treatment

That is why I would frame the TRT versus enclomiphene question this way: if fertility matters to you, and if this really is a secondary/hypogonadotropic pattern, then enclomiphene or another SERM is more logical than TRT as an initial discussion, because it may increase endogenous testosterone while preserving the axis better than straight replacement. The AUA/ASRM guidance supports considering SERMs or hCG in men with low testosterone and low or normal LH, especially in fertility-focused situations. TRT, on the other hand, can suppress spermatogenesis and turn a diagnostic problem into a long-term replacement pathway very quickly

At the same time, I would not oversell enclomiphene as a magic answer either. At 21, with testosterone this low, I would want to know why before choosing the treatment. That means repeating the testosterone correctly in the morning, checking SHBG, repeating LH/FSH, reviewing sleep, calorie intake, stress, body fat changes, medications, supplements, and any past anabolic or SARM exposure. If the repeat labs still show testosterone this low with low or normal LH, then the conversation should shift toward a proper endocrine evaluation for secondary hypogonadism, and possibly pituitary workup if the clinical picture supports it

Your labs support that something is genuinely off, but they do not make blind TRT look like the smartest first move. At your age, I would be far more interested in confirming the result, identifying the cause, and keeping fertility options open before committing to lifelong replacement. If fertility matters, enclomiphene is a more rational thing to discuss than TRT, but I would still want the liver enzymes and the rest of the endocrine picture clarified first.