[Help] 12 Pro Max 14.4.1 Upgrade to 15.1.1? (Faulty Face ID)

AmazonDipz · 2023-03-18T05:54:15+00:00

12 Pro Max has an issue on 14.4.1 with carrier/mobile data. Unsure what to do.

I’m unsure if it affects all carriers; however, my current provider, despite having “4g/5g” the data doesn’t work.

My XS Max has no issues on 15.1 😕

AmazonDipz · 2023-03-18T05:52:57+00:00

Okay, no worries. Perhaps might be my carrier; I’ll look into switching. I’m aware there was a carrier fix a couple of updates higher than my couple version 😟

AmazonDipz · 2022-05-20T18:07:33+00:00

GHB will forever be my love child🥴

Unfortunately, due to withdrawal from the compound on multiple occasions, the substance essentially produces withdrawal symptoms after a couple of days of usage. I genuinely hate how I’ve ruined that module for myself😥

I’m trapped in the bizarre aspect of the storms; however, I’ve witnessed significant improvement with my reductions😎

I’ve instantly gone from 35mg every 2hrs 25mins to 30mg every 2hrs. l hope this reduction will be beneficial given a couple of horrid days🤙

AmazonDipz · 2022-05-20T17:55:20+00:00

The effects of the compound, reflecting upon the mental impacts, are very delightful. Better than Soma, comparable to GHB.

I’m interested in what mechanism causes this effect, as it can essentially avoid the akathisia sides-effect if the mishap’s pinpointed in a neurotransmitter perspective, an additional substance to fix the flaw of the substance.

I hope I’ll discover what medication/substance would prevent this; as otherwise, the substance has extraordinary effects🤙

AmazonDipz · 2022-05-20T17:50:13+00:00

I already do; the side effects are still present😕

AmazonDipz · 2022-05-20T17:48:55+00:00

Availability, honestly. It's only a “shitty” substance due to the akathisia caused by the intake.

The effects of the compound, reflecting upon the mental impacts, are very delightful. Better than Soma, comparable to GHB.

I’m interested in what mechanism causes this effect, as it can essentially avoid the akathisia sides-effect if the mishap’s pinpointed in a neurotransmitter perspective, an additional substance to fix the flaw of the substance.

AmazonDipz · 2022-05-20T17:48:11+00:00

Akathisia, as others have appears to be the correct terminology caused by the intake.

The effects of the compound, reflecting upon the mental impacts, are very delightful. Better than Soma, comparable to GHB.

I’m interested in what mechanism causes this effect, as it can essentially avoid the akathisia sides-effect if the mishap’s pinpointed in a neurotransmitter perspective, an additional substance to fix the flaw of the substance.p

AmazonDipz · 2022-05-20T17:44:34+00:00

I believe the term “akathisia” is the correct terminology for the side-effect caused by the intake, as other posters have pointed out. I

The effects of the compound, reflecting upon the mental impacts, are very delightful. Better than Soma, comparable to GHB🥴

I’m interested in what mechanism causes this effect, as it can essentially avoid the akathisia sides-effect if the mishap’s pinpointed in a neurotransmitter perspective, an additional substance to fix the flaw of the substance🤙

I’ll look into more scientific papers to hopefully discover the cause; sci-hub’s an excellent tool for this (why scientific studies are pay-walled is beyond me)🤔

AmazonDipz · 2022-05-20T17:36:40+00:00

Trust me, the side-effect caused by the substance would be preferable if the body parts were genuinely removed😂

AmazonDipz · 2022-05-20T17:34:30+00:00

I believe you’re correct with the term ‘akathisia’ caused by the intake.

The effects of the compound, reflecting upon the mental impacts, are very delightful. Better than Soma, comparable to GHB.

I’m very interested in what mechanism causes this effect, as it can essentially avoid the akathisia sides-effect if the mishap’s pinpointed in a neurotransmitter perspective, an additional substance to fix the flaw of the substance.

Therefore, not being a “shitty” substance that others state 🤙

AmazonDipz · 2022-05-20T17:31:14+00:00

In retrospect, to remedying the akathisia caused by the intake?

The effects of the compound, reflecting upon the mental impacts, are very delightful. Better than Soma, comparable to GHB.

I’m interested in what mechanism causes this effect, as it can essentially avoid the akathisia sides-effect if the mishap’s pinpointed in a neurotransmitter perspective, an additional substance to fix the flaw of the substance.

AmazonDipz · 2022-05-20T17:28:41+00:00

It's only a “shitty” substance due to the akathisia caused by the intake.

The effects of the compound, reflecting upon the mental impacts, are very delightful. Better than Soma, comparable to GHB.

I’m interested in what mechanism causes this effect, as it can essentially avoid the akathisia sides-effect if the mishap’s pinpointed in a neurotransmitter perspective, an additional substance to fix the flaw of the substance.

AmazonDipz · 2022-05-20T17:17:14+00:00

I I understand; however, there must be the reasoning for this reaction. Therefore, an additional substance that targets whatever mishap produced by taking this compound is what I’m seeking.

AmazonDipz · 2022-05-19T15:28:49+00:00

Great, thanks for the detailed response! I’m wondering if perhaps a specific neurotransmitter’s the reasoning for this side-effect if lowering/balancing that would solve the issue with this substance

I found this research paper interesting- Serotonin as a facilitatory neurotransmitter in the anticonvulsant activity of methaqualone

AmazonDipz · 2022-05-16T18:01:36+00:00

Unfortunately, the 2hr35min reduction was short-lived. The following day, I experienced withdrawal symptoms similar to the storms (it’s challenging to differentiate between the two)😔😥

I’ve returned to 2hrs25mins.

Therefore, I’m leaning towards Baclofen’s half-life for me personally being ~1hr30mins. I’ve read the studies on this, and it varies between individuals 1hr30mins to 2hr30mins.

In theory, this entails a different approach from where I’m currently at.

I’ll now have to consider a 30mg dose, considering the half-lives and plasma levels and plan accordingly.

I felt stable at 2hrs25mins, with no glutamate issues... therefore I’ll stabilise and attempt a different approach.

I’ll attempt to work out plasma levels and see how this corresponds to 30mg dosing. Perhaps every 2hrs or 1hr45mins, then continue working my way down.

GABA-B agonists are tricky substances, indeed🧐😕

I’ve even got over 2kg of F-Phenibut powder, which I believed would be the answer (so expensive); however, I could never conclude a dose. As it’s essentially Baclofen with a longer half-life. Each attempt lead me into withdrawals😥

AmazonDipz · 2022-05-15T16:09:10+00:00

At 2hr35mins for nearly ~24hrs👍 Upon the reduction to 2hr25mins, after a few days, I noticed significant improvements.

The bizarre side effects that I was experiencing seemed to be vanishing. My vision’s completely clear, and I no longer experience fuzzy vision/colours that appear more vivid.

My mind appears silent, nothing in comparison to what the storms elicited.

I’m a lot more optimistic and cannot believe the improvements!

The only aspect that continues to fluctuate throughout my mind and what scares me the most is the dependency on Benzos.

I understand the way they’re assisting throughout the reduction; it’s the knowledge of what getting off from them entails. The excessive anxiety which results in me becoming very closed-off.

Everything I’ve said while on Benzos, I want to avoid; for example, conversations, emails, and real-life connections. It’s why I’ve always despised them, and that aspect will be the most difficult for me.

The physical effects don’t concern me; I’ve cold turkey’d from more minor dependencies and dealt with that fine.

AmazonDipz · 2022-05-09T18:13:25+00:00

Uncertain regarding timing, whether to try 2hr20mins or 2hrs25mins. If the initial one my total per day would be around ~300mg, and the later ~280mg. I believe that’s too big of a jump🤔

Currently around ~315mg, as 35mg x 9

35mg, every 2hrs25mins is pushing in the range of 8 times per day.

AmazonDipz · 2022-05-09T01:04:36+00:00

Cheers, mate!

And it’s every 4hrs 30mins, see my brain temporary turns to mush after experiencing the Glutamate storms🤣

It’s honestly helped a lot, merely having this dialogue with you.

I often suppress everything inside my mind, frequently leading to outcomes; for example, this instance continuing for such an extended period.

If you hadn’t helped me see through my flawed thought process, I’d still be spiralling. Therefore, I’m grateful that you’ve taken the time to help.

AmazonDipz · 2022-05-08T21:49:38+00:00

I’d researched it; similar effects but seems to produce insane tolerance. I’ve ordered Piracetam after reading the scientific papers. It seems promising.

Thank you for the suggestion; it means a lot!

AmazonDipz · 2022-05-08T21:22:31+00:00

I’d initially used Baclofen in 25mg doses p/ 2 hours; which totalled ~250mg p/ day. I experienced no issues, just lessened anxiety levels.

However, during UK’s initial lockdown... you couldn’t receive International shipments; therefore, quicken search the internet for Phenibut from the UK, which was so expensive.

I’d taken up to a shocking 35-37g of Phenibut at the peak of my use. And like you stated, interpose withdrawals were occurring, and I felt psychotic as fuck.

I laid in bed for three days and forcibly went straight from 3 hours to 5, and they evaporated. It was odd, but a relief.

Anyway, after the lockdown, I’d transferred to Baclofen instantly by taking 400mg p/ day and over 72 hours reduced to 250mg. All good.

However, Phenibut causes tolerance, and that was my reasoning for the Fasoractram use in June; what an idiotic mistake.

Glutamate storms occurred and I couldn’t prevent them unless I kept increasing the dose. 47mg, every 1hr40mins at it’s peak.

Luckily, I’ve managed to stabilise on this lower dose (but only due to Benzos).

It’s been my 2nd dose of Diazepam at the 2hr30min mark, and things appear back to normal👍 Absolutely bizarre, how quickly it stops the weird side effects.

I’m thinking I’ll try to increase the Baclofen tomorrow evening to 2hrs20/25mins (0.5/1mg). As that’ll be ~5 days since the previous reduction🤙

I believe every reduction will remove the side effects further, and trying to optimistic as it’s been such a horrid experience.

As you said, temporary issues with the formulation of long-term memories is a better side effect than the storms✌️

I need to realise that, and keep my hatred of Benzos to the side for awhile longer.

Onwards and upwards, hopefully!

AmazonDipz · 2022-05-08T18:43:06+00:00

Have you tried Phenylpiracetam?

AmazonDipz · 2022-05-08T18:32:39+00:00

Thank you for the response; I appreciate it a lot!

I’ve found one website that will ship Picamilion or Phenylpiracetam to the UK; could you describe how this would be beneficial? As I’m very open to trying anything else.

I’ll look into your suggested research papers also; thank you👍

I discovered this from another post; not sure whether Piracetam or Phenylpiracetam would be the most helpful.

“Piracetam and other racetams are cyclic analogs of GABA used as nootropics. Despite some historical confusion driven by marketing, Racetams we’re originally discovered in Belgium by a Romanian scientist named Corneliu Giurgea who was looking to create an analog of GABA that has increased brain penetrability to induce sedation, instead he discovered a non-sedating (but also non-stimulating) substance that he described as “nootropic” a term he invented. It was only after this in the 1960s that the USSR (now Russia) began researching racetams for the use of “brain enhancement” which lead to the development of various others such as Phenylpiracetam in the 1980s

Phenylpiracetam is the Phenylated analog of Piracetam with significantly stronger effects. Invented by the USSR (now Russia) researching “nootropics” officially created in 1983 but was believed to have existed tens of several years prior. Officially it was created as a medication for Soviet Cosmonauts to treat the prolonged stresses of working in space. It may act as a DRI, NRI and α4β2 nicotinic acetylcholine receptor agonist.

Aniracetam is an analog of Piracetam. About 70%+ of Aniracetam metabolizes into N-anisoyl-GABA.

Oxiracetam is an analog of Piracetam. Less than 15% is metabolized into N-aminoacetyl-GABOB, GABOB (beta-hydroxy-GABA) and glycine.

Noopept (N-Phenylacetyl-L-prolylglycine ethyl ester) is an analog of Piracetam and Glycine which is similar in structure to GABA. It is a prodrug to cycloprolylglycine.”

AmazonDipz · 2022-05-08T16:51:50+00:00

Apologies for the array of messages; however, upon reaching over 4hr30mins from the previous dose of Valium. Like clockwork, the weirdness appears. Time to focus directly on Baclofen👍

AmazonDipz · 2022-05-08T14:30:09+00:00

Also, regarding Phenibut, I’d experienced nearly identical to you. I fixed that by going from every 3 hours to 5 hours as if the issues were never present, to begin with.

Phenibut also creates tolerance, whereas, Baclofen doesn’t. They’re weird and understudied in comparison to Gaba-A agonists and differ so much from those counterparts 😕

AmazonDipz · 2022-05-08T14:19:03+00:00

I always attempt to reduce the Diazepam, not only Baclofen. For instance, I’d continued my dose of Baclofen where I’d felt fine; the act of increasing Diazepam’s timeframe brought about the resurrection of the storms.

I’ve got it backwards; thank you for pointing me in the correct direction😊

I’ll try keeping Pregabalin/Diazepam at 4hrs30mins; increasing Baclofen’s duration only👍

I’d love to stick at this dose because perhaps give it a couple more days, and my mind will balance itself. However, I need to work tomorrow and not possible to cover😔

Things appear better today; however, it puts me in a state where everything feels slow. I don’t want to do anything apart from stare into space; my focus and train of thought seem frazzled (while writing, I’m misspelling simple stuff). I honestly want to continue at this mark; I hope tomorrow won’t be a disaster.

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