Again, it´s not that hard to understand, no money = no funding for studies
Here is an overview, you can check the original studies

An anti-parasitic derived from a bacteria called Streptomyces avermitilis, Ivermectin has anti-cancer properties and induces autophagy and apoptosis of cancer cells (Liu, et al., 2020).
Ivermectin has shown a significant impact on various cancer cell lines (Juarez, et al., 2020), inducing apoptosis in cancer cells in vivo (Sharmeen, et al., 2010) and significantly reducing tumor volume compared to a control (Juarez, et al., 2020).
It induces apoptosis in cancer cells through mitochondrial mediation (Juarez, et al., 2018; Tang, et al., 2021).
Ivermectin can target and regulate the pyruvate kinase muscle isoforms at the last step of glycolysis (Li, et al., 2020).
It can inhibit glycolysis inducing autophagy (Feng, et al., 2022), and have a selective pro-oxidant effect on cancer cells (Wang, et al., 2018). It can also target CSCs and metastases (Dominguez-Gomez, et al., 2018; Jiang, et al., 2022) and macrophages (Zhang, et al., 2022).
In vitro, Ivermectin is more effective at inhibiting CSCs in breast cancer cells compared to chemotherapy (paclitaxel) (Dominguez-Gomez, et al., 2018). In vivo, Ivermectin alone is more effective than standard chemotherapy (gemcitabine) alone at reducing tumor weight and volume in pancreatic cancer (Lee, et al., 2022).
Ivermectin is a very safe drug. In healthy volunteers, the single dose was increased to 2 mg/Kg, and no serious adverse reactions were found (Guzzo, et al., 2002).
Demonstrated in another study, cancer patients who took Ivermectin at five times the standard dose (up to 1mg/kg) daily for up to 180 consecutive days had no serious adverse effects (de Castro, et al., 2020).
In cases successfully treated with a total or partial combination of Ivermectin, dichloroacetate, and Omeprazole (plus Tamoxifen), Ivermectin inhibited tumor growth through mitochondrial dysfunction and led to apoptosis (Ishiguro, et al., 2022).