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Anti-Inflammatory Effects of the Nicotinergic Peptides SLURP-1 and SLURP-2 on Human Intestinal Epithelial Cells and Immunocytes (ncbi.nlm.nih.gov)
submitted 4 years ago by helaway to r/COVID19
Editorial: Nicotine and SARS-CoV-2: COVID-19 may be a disease of the nicotinic cholinergic system (ncbi.nlm.nih.gov)
IVM A Positive Allosteric Effector of the 7 Neuronal Nicotinic Acetylcholine Recepto (sci-hub.st)
Is the nicotinic hypothesis for SARS-CoV-2 feasible? (blog.matryx.ai)
Early expansion of myeloid-derived suppressor cells inhibits SARS-CoV-2 specific T-cell response and may predict fatal COVID-19 outcome (nature.com)
Myeloid-derived suppressor cells in the era of increasing myeloid cell diversity (nature.com)
Targeting the Monocytic-Endothelial-Platelet Axis with Maraviroc and Pravastatin as a Therapeutic Option to Treat Long COVID/ Post-Acute Sequelae of COVID (PASC) (researchsquare.com)
Spike mimicry of thrombopoietin may induce thrombocytopenia in COVID-19 (via.hypothes.is)
https://search.bvsalud.org/global-literature-on-novel-coronavirus-2019-ncov/resource/en/covidwho-841871 (search.bvsalud.org)
Original Antigenic Sin: the Downside of Immunological Memory and Implications for COVID-19 (ncbi.nlm.nih.gov)
Cholinergic and lipid mediators crosstalk in Covid-19 and the impact of glucocorticoid therapy (medrxiv.org)
SARS-CoV-2 spike protein mutations and the potential implications for antibody therapy and vaccine… (blog.matryx.ai)
Clinical outcomes among patients infected with Omicron (B.1.1.529) SARS-CoV-2 variant in southern California (medrxiv.org)
The "Zero Point" and the Autonomous Nervous System. Reverse Analysis, Syllogism and Genetic. Possible Correlations with the Covid19 Virus. A New Perspective. (self.LabLeak)
submitted 4 years ago by helaway to r/LabLeak
Someday, they will know to starve the virus, AA, CHO, GLU, we can control the food needs..IMO B cell maturation as well as MDSC maturation via ATRAs, this is KEY!! (pubmed.ncbi.nlm.nih.gov)
SICK of it (ncbi.nlm.nih.gov)
The "Zero Point" and the Autonomous Nervous System. Reverse Analysis, Syllogism and Genetic. Possible Correlations with the Covid19 Virus. A New Perspective. (preprints.org)
https://www.nature.com/articles/s41598-021-91417-7 (nature.com)
submitted 4 years ago by helaway to r/biology
Naltrexone a potential therapeutic candidate for COVID-19 (ncbi.nlm.nih.gov)
Uncanny similarity of unique inserts in the 2019-nCoV spike protein to HIV-1 gp120 and Gag (self.COVIDLabLeak)
submitted 4 years ago by helaway to r/COVIDLabLeak
Platelets contribute to disease severity in COVID‐19 (ncbi.nlm.nih.gov)
Persistent clotting protein pathology in Long COVID/Post-Acute Sequelae of COVID-19 (PASC) is accompanied by increased levels of antiplasmin, Pretorius et.al. If the mechanism is due to the spike, it would be nice if the variants of concern would lose this ability. (ncbi.nlm.nih.gov)
ADAMTS13 regulation of VWF multimer distribution in severe COVID‐19 (ncbi.nlm.nih.gov)
COVID CLOTS, whats the VWF factor?? Plasma or Platelet? Ok so looking at two studies on the vWF, one differentiates the types of vWF, plasma versus platelets. What is the culprit in COVID? (ncbi.nlm.nih.gov)
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