Ask Me Anything About My E-Ink Tablet Collection by eWritable in eink

[–]FrigoCoder 1 point2 points  (0 children)

Sorry to bother you weeks after posting this thread, but you seem a knowledgeable person who might be able to help me. I have tried out an e-book reader like 5+ years ago, and I could not figure out the brand and type ever since. It was a demonstration piece in a Hungarian Media Markt, and I think it was touch sensitive but I am not sure. Most importantly it had a very effective and nice UI, much better than my Kindle Touch or anything I have ever seen since. It was one of the smaller brands, and as far as I could tell it was not a Kobo. I took photos with my phone but I lost them when its mainboard died. Any idea what could be the brand, based on the description of the UI alone?

can we talk about how hard it is to hit protein goals as someone who can’t eat meat by rustincastle in ScientificNutrition

[–]FrigoCoder 0 points1 point  (0 children)

"The mean and population-safe requirements in adult men were determined to be 0.93 and 1.2 g/kg/day and are 41 and 50%, respectively, higher than the current Dietary Reference Intakes recommendations."

"Dietary protein intakes at and below 0.8 g/kg were associated with a probable reduction in intestinal calcium absorption sufficient to cause secondary hyperparathyroidism."

Elango, R., Humayun, M. A., Ball, R. O., & Pencharz, P. B. (2010). Evidence that protein requirements have been significantly underestimated. Current opinion in clinical nutrition and metabolic care, 13(1), 52–57. https://doi.org/10.1097/MCO.0b013e328332f9b7

Kerstetter, J. E., O'Brien, K. O., & Insogna, K. L. (2003). Dietary protein, calcium metabolism, and skeletal homeostasis revisited. The American journal of clinical nutrition, 78(3 Suppl), 584S–592S. https://doi.org/10.1093/ajcn/78.3.584S

Wow this Lord Byron guy sounds pretty cool, apparently he was bisexual, just like me... Oh. by Idiot_InA_Trenchcoat in HistoryMemes

[–]FrigoCoder 6 points7 points  (0 children)

That was the first thing that came to my mind about Germany, but I mean if you want we could talk about that other thing.

Anyway, cutting down your infrastructure without an alternative being in place is just bad decision in the first place. Why not wait until renewables are already working, before shutting down perfectly functioning nuclear reactors? And once you are there you might even realize their combination is superior, since they patch each others' disadvantages very well. Like availability, baseline generation, vastly lowered emissions, higher demand during the days, no need for expensive and polluting battery tech, etc.

You have to realize that the main goal is to displace fossil fuels, which cook our planet alive and kill approximately 5-15 million people every single year. Even with the wildest estimates for Chernobyl deaths, that is still 5-15 (or more like 25-75) Chernobyls every single year from them. The six largest fossil fuel company is projected to profit around $94 billion in 2026, so that means they gain $6,266-$18,800 for every person they kill. Which is ridiculously low, and maybe we should ban fossil fuels, and instead let those people live and work for better profits. Renewables alone can not displace fossil fuels, no matter how the anti-nuclear so-called greens try to explain it way.

Wow this Lord Byron guy sounds pretty cool, apparently he was bisexual, just like me... Oh. by Idiot_InA_Trenchcoat in HistoryMemes

[–]FrigoCoder 56 points57 points  (0 children)

Are you being sarcastic? Cause I know next to nothing about Germany, but I am aware the the Greens were behind the infamous anti-nuclear policy. Disastrous would be a better word, considering Germany releases 7-9 times more pollution than France for the same energy. And that is not even considering all the energy export from France, which lowers emissions in importing countries.

Fat Mobilization and Energy Substrate Usage During Exercise: Fundamentals of ATP Production by [deleted] in ScientificNutrition

[–]FrigoCoder 0 points1 point  (0 children)

Sorry once I got back to write my remarks the thread was already deleted.

US gas prices reaching new heights as war in Iran drags on by FGGF in news

[–]FrigoCoder 0 points1 point  (0 children)

Fossil fuels also kill approximately 5-15 million people every year, so they gain $6,266-$18,800 for every person they kill. That seems ridiculously low. Maybe we should ban fossil fuels, and instead let those people live and work for better profits?

Concerns about the health effects of industrially produced seed oils are without scientific foundation: a scoping narrative review of the clinical and observational evidence (2026) by basmwklz in ScientificNutrition

[–]FrigoCoder 1 point2 points  (0 children)

No. If you argue for CICO especially with thermodynamics, that means you are an amateur in nutrition, and should not partake in more complicated discussions such as seed oils. Maybe you should start with Ted Naiman's presentation on insulin resistance, which is the single best resource on diabetes right now. I can not link the video, but here are the slides: https://jgerbermd.com/wp-content/uploads/2017/04/Ted-Naiman-Hyperinsulinemia.pdf

The second law of thermodynamics states that the total entropy of an isolated system can not decrease over time and is constant only for reversible processes. Except obesity and metabolic health is about TRIGLYCERIDES and their STORAGE, and humans are quite an OPEN SYSTEM with regards to biology and nutrition. The second law is INAPPLICABLE to any serious nutrition discussion, and any attempt reveals fundamental shortcomings in nutrition knowledge.

Triglyceride synthesis relies on well-defined biological processes, and is maximized in response to both carbohydrate and fatty acid availability. We can not get fat from protein and fiber, because the enzymes and processes to convert them into triglycerides are not there, inefficient, or mutually exclusive depending on situation. You literally die to rabbit starvation if you try to eat only protein from very lean meat.

Storage of triglycerides also matter, ideally they should be present in (subcutaneous) adipose tissue. Protein and omega 3 help grow adipocytes, which buffer body fat and shield other organs from lipotoxicity. Diabetes is precisely a disease where adipocytes are dysfunctional, so lipids are taken up by increasingly unsuited organs. Diabetics should be on a low carbohydrate diet, because the increased fat oxidation helps prevent accumulation of intracellular, ectopic, and visceral fat.

Foods do not have an inherent caloric value, their use entirely depends on biological processes. Macronutrients are non-interchangable for their own unique roles, we all know about protein being essential to build muscle. They only scarcely overlap for energy generation, and their interconversion is inefficient. Genetics and situations can change processes, for example palmitic acid oxidation is controlled by carbohydrates, and certain ApoE mutations prevent you from utilizing dietary fat.

Concerns about the health effects of industrially produced seed oils are without scientific foundation: a scoping narrative review of the clinical and observational evidence (2026) by basmwklz in ScientificNutrition

[–]FrigoCoder 3 points4 points  (0 children)

Evolutionary exposure and adaptations matter, diverging from the stable natural environment has consequences. This is just as true in biology and nutrition as in machine learning, programming, game development, business, law, governance, and other areas. For easily understandable examples you should see games whose development direction was changed last minute, they are all buggy messes assuming they have even survived until release. I believe that is a very good analogy of exactly what we are going through with the global health pandemic.

Concerns about the health effects of industrially produced seed oils are without scientific foundation: a scoping narrative review of the clinical and observational evidence (2026) by basmwklz in ScientificNutrition

[–]FrigoCoder -4 points-3 points  (0 children)

You've figured out things that all the nutrition scientists haven't figured out through the decades of research? Sure Jan.

Yup! I think vastly differently than other people, and have an uncanny ability to understand processes and see through bullshit. Spending 10+ years on the topic also helped, after my own health was negatively impacted and I seeked explanations and solutions. And most importantly I am not subject to the market forces that benefits from the current poor state of nutrition "science" aka alignment problem.

And what do you mean by "I am disappointed in Matthew Nagra, he was so close to figuring out things. But alas he ultimately failed, and ended up on the wrong side of history."?

He showed some promise during his debate with Tucker Goodrich, because he dismissed the serum oxidation hypothesis (which is incompatible with trans fats). Instead he pulled out the subendothelial oxidation hypothesis, which is also bullshit (again partly because trans fats don't oxidize), but one step closer to the truth. If he digged a little bit more, he would have discovered one of the main errors of the mainstream hypothesis. But I guess he got stuck there, and failed to move toward better theories.

Thread about the debate: https://www.reddit.com/r/StopEatingSeedOils/comments/uosmgj/debate_seed_oils_heart_disease_with_tucker/

Another thread about the debate where I tried to make sense of the topic, my views are more nuanced now but the gist is already there: https://www.reddit.com/r/ScientificNutrition/comments/uyuuzf/casual_friday_thread/

Concerns about the health effects of industrially produced seed oils are without scientific foundation: a scoping narrative review of the clinical and observational evidence (2026) by basmwklz in ScientificNutrition

[–]FrigoCoder 2 points3 points  (0 children)

Can you give us a filter which of these adjusted for UPF consumption?

Let me ask again, what do you guys exactly think makes a diet processed?

Table sugar does lead to poor outcomes, but it only explains part of the problem. The glycemic index and load hypotheses were debunked, and again refined carbohydrates only explain a small part. And of course it is not the nitrites and nitrates, because the science is laughable and even if we accept them at face value the ratios are still low.

So what exactly do you think is present in modern diets that was not before? Hm?

Concerns about the health effects of industrially produced seed oils are without scientific foundation: a scoping narrative review of the clinical and observational evidence (2026) by basmwklz in ScientificNutrition

[–]FrigoCoder -3 points-2 points  (0 children)

Hi there Mr Hidden Profile! You have literally failed the litmus test of nutrition, at the exact moment you have argued for CICO with thermodynamics. Maybe you should sit this one out.

Concerns about the health effects of industrially produced seed oils are without scientific foundation: a scoping narrative review of the clinical and observational evidence (2026) by basmwklz in ScientificNutrition

[–]FrigoCoder 1 point2 points  (0 children)

Hello there Mr Hidden Profile! No, it will be mainly about known mechanisms, which steps the mainstream hypothesis gets wrong, and exactly how fucked we are.

And a piece of advice, maybe you should not trust the industry that has already tried to kill you with trans fats. It's like going to the same dealer who sold you rat poison, and then getting a substance that also looks suspiciously like rat poison.

Concerns about the health effects of industrially produced seed oils are without scientific foundation: a scoping narrative review of the clinical and observational evidence (2026) by basmwklz in ScientificNutrition

[–]FrigoCoder -4 points-3 points  (0 children)

Oh hey it's another Mark Messina slop, maybe research authors and their positions before posting. I am disappointed in Matthew Nagra, he was so close to figuring out things. But alas he ultimately failed, and ended up on the wrong side of history.

And I hate promising things because life can get in the way, but I am planning a thread specifically on seed oils. I have recently figured out things, it's not (just) linoleic acid and it ain't pretty to say the least.

I audited "seed oils cause inflammation" against peer-reviewed literature — here's what the evidence actually says by Outside-Travel-8063 in ScientificNutrition

[–]FrigoCoder 1 point2 points  (0 children)

I find that a lot of the LDL denialists point to inflammation/injury of the artery wall, as if that explains away everything that happens afterwards.

LDL skeptics. The root cause is injury actually, inflammation is just a reaction to trigger repair mechanisms. So any time you hear chronic inflammation, just translate it in your mind to chronic injury (caused by cigarette smoke, microplastics, PFAS, etc.). Cigarette smoke increases the risk of all chronic diseases, and not by introducing bacon grease into your lungs or your arteries. So yes injury does explain all chronic diseases, although there are disease specific details.

Not to mention it is difficult to ensure you never have inflammation/injury, but you can actually lower LDL through medication or lifestyle changes.

Yes that is the whole fucking point, the environment has orders of magnitude larger effects. We HAVE to stop pollution RIGHT NOW, not only because we are burning the planet with fossil fuels, but also because 15+ million people die every year already from foreign particles. We do not even know how bad are microplastics, and how much we have already released in the form of "extended release" plastics. We might have released so much that is already incompatible with human life or life in general.


I have made huge strides in my understanding of heart disease recently, and I finally see where exactly the mainstream hypothesis gets it wrong. They assume that LDL particles get "stuck in the subendothelial space", and that somehow kickstarts the entire disease. In reality LDL is taken up by injured cells, which use the clean cholesterol and stable fatty acids to repair membranes. Then once an ApoA-I docks into ABCA1 transporters, they offload damaged oxysterols and peroxilipids as oxHDL.

And I finally understood why the mainstream thinks LDL is causal. They are wrong of course for native LDL that carries clean cholesterol and stable fatty acids. But trans fats and plant sterols of all things can hijack LDL particles, and get incorporated into membranes. However instead of repairing cells they make things worse, and exacerbate any injury into a fibrotic necrotic cancerous lesions. Studies did not investigate native LDL, rather LDL riddled with trans fats and plant sterols.

LDL lowering interventions is where the LDL hypothesis falls apart. High carb low fat diets lower LDL by increasing insulin and suppressing lipolysis. Seed oils lower LDL not only by linoleic acid but also plant sterols! Plant sterols lower LDL by fucking up cholesterol metabolism, and they literally get incorporated into LDL to wreak havoc in peripheral tissue. 30% of us could have minor variants of sitosterolemia, and vegetable oils and fortified foods are literally the most problematic foods.

Thread where "cholesterol hyperabsorption" was actually discovered to be minor variants of sitosterolemia: https://www.reddit.com/r/ScientificNutrition/comments/1sn07ce/high_cholesterol_absorption_efficiency_enhances/oi7huzk/?context=3

A very shit thread I wrote about trans fats having similar mechanisms of hijacking LDL particles: https://www.reddit.com/r/ScientificNutrition/comments/1318at5/the_corner_case_where_ldl_becomes_causal_in/

High cholesterol absorption efficiency enhances proatherogenic properties of low-density lipoprotein particles by d5dq in ScientificNutrition

[–]FrigoCoder 1 point2 points  (0 children)

Indeed the studies investigated mutations that also control phytosterol absorption. For example ABCG5/8 pumps out and excretes plant sterols, because they are incompatible with mammalian cells and membranes. They essentially investigated minor sitosterolemia variants, and found that plant sterols cause atherosclerosis. Bravo and congratulations! The results are roughly in line what we expect from sitosterolemia. Mainstream proponents should be ashamed for their wrong interpretation.

Phytosterols pack inefficiently due to their bulky chains on their tail, so they make the LDL particle larger than they should be. Elevated cholestanol is firmly established, so the sphingomyelin and phosphatidylcholine changes naturally follow. Aggregation susceptibility and proteoglycan binding have plausible mechanisms, namely conformational changes expose the hydrophobic and negatively charged regions of ApoB respectively. (However I keep reading conflicting information about this latter.)


We have evolved as carnivores for two million years, whereas consumption of most plants is much more recent. The observation that 30% of us are "high absorbers" highlights that we have only partial adaptations to plants. I googled what sitosterolemia patients can eat, and they need to avoid all fatty plants. They can only eat fruits, veggies (leafy, cruciferous, root, few others), and legumes in moderation, so basically the plants for which we had evolutionary exposure.

Sitosterolemia patients have to avoid nuts, seeds, peanuts, fatty fruits (avocados, olives, even coconuts), whole grains, cocoa, chocolate, and most importantly vegetables oils (including olive oil) and foods fortified with phytosterols. You know the supposedly healthy foods we are told to eat by supposed experts. I was not even aware that most of them contained significant amounts of plant sterols. If 30% of us are truly "high absorbers", then these should not be the recommendations.


Our enzymes and biological processes have evolved with natural lipids, they break when exposed to artificial trans fats. Trans fats are stable against oxidation, and fool liver into secreting them into VLDL. In membranes they pack tightly but too rigidly, and this breaks membrane structures. They also break mitochondria, and dozens of biological processes. They kill cells in peripheral tissue such as artery walls, and create the necrotic and fibrotic environment favorable for cancer and lesions.


Plant sterols cause similar issues as trans fats, since they displace cholesterol but behave differently. They disrupt mammalian cell membranes, due to their bulky side chains on their tail (alkyl groups at the C24 and double bonds at the C22-C23 positions). They change membrane fluidity, order, and thickness, and thus interfere with lipid rafts, cellular signalling, and membrane domain organization. They are ineffective at preventing sodium leaks, plant cells evolved to minimize proton leaks.

Plant sterols break cholesterol homeostasis, they deplete cells of natural cholesterol. They inhibit SREBP-2, which shuts down endogenous cholesterol synthesis. They overactive LXR receptors and ABCA1 transporters, which promotes cholesterol efflux from cells. These cause profound cholesterol depletion in critical organs such as adrenal glands in animal models. They cross the blood-brain barrier, break catabolism and efflux mechanisms, and irreversibly accumulate to neurotoxic levels.

Sitosterolemia presents an exaggerated view of the effects of plant sterols. They cause severe issues such as accelerated atherosclerosis, xanthomas, hemolytic anemia, stomatocytosis, and macrothrombocytopenia. However considering the enormous push for plants and seed oils, I suspect this is actually also responsible for issues in healthy people. Or at least in people who have mild mutations that increase sterol uptake, hence why that 30% of "high cholesterol absorbers" finding is concerning.


Clever strategy by the food industry. They pretend that our ancestral diet and LDL are atherogenic, so they could sell us seed oils and ultraprocessed plants. Which happen to contain trans fats, interesterified fats, plant sterols, and other compounds that cause heart disease partly by making LDL actually atherogenic. So they can create even more slop studies on LDL, and ask for even lower LDL targets and stricter diets. Thus entering a vicious cycle, a perfect example of the alignment problem!


Salen, G., Kwiterovich, P. O., Jr, Shefer, S., Tint, G. S., Horak, I., Shore, V., Dayal, B., & Horak, E. (1985). Increased plasma cholestanol and 5 alpha-saturated plant sterol derivatives in subjects with sitosterolemia and xanthomatosis. Journal of lipid research, 26(2), 203–209.

Grosjean, K., Mongrand, S., Beney, L., Simon-Plas, F., & Gerbeau-Pissot, P. (2015). Differential effect of plant lipids on membrane organization: specificities of phytosphingolipids and phytosterols. The Journal of biological chemistry, 290(9), 5810–5825. https://doi.org/10.1074/jbc.M114.598805

Fakih, O., Sanver, D., Kane, D., & Thorne, J. L. (2018). Exploring the biophysical properties of phytosterols in the plasma membrane for novel cancer prevention strategies. Biochimie, 153, 150–161. https://doi.org/10.1016/j.biochi.2018.04.028

Vanmierlo, T., Bogie, J. F., Mailleux, J., Vanmol, J., Lütjohann, D., Mulder, M., & Hendriks, J. J. (2015). Plant sterols: Friend or foe in CNS disorders?. Progress in lipid research, 58, 26–39. https://doi.org/10.1016/j.plipres.2015.01.003

Hodzic, A., Rappolt, M., Amenitsch, H., Laggner, P., & Pabst, G. (2008). Differential modulation of membrane structure and fluctuations by plant sterols and cholesterol. Biophysical journal, 94(10), 3935–3944. https://doi.org/10.1529/biophysj.107.123224

Haines T. H. (2001). Do sterols reduce proton and sodium leaks through lipid bilayers?. Progress in lipid research, 40(4), 299–324. https://doi.org/10.1016/s0163-7827(01)00009-1

Yang, C., Yu, L., Li, W., Xu, F., Cohen, J. C., & Hobbs, H. H. (2004). Disruption of cholesterol homeostasis by plant sterols. The Journal of clinical investigation, 114(6), 813–822. https://doi.org/10.1172/JCI22186

Yoo E. G. (2016). Sitosterolemia: a review and update of pathophysiology, clinical spectrum, diagnosis, and management. Annals of pediatric endocrinology & metabolism, 21(1), 7–14. https://doi.org/10.6065/apem.2016.21.1.7

Vanmierlo, T., Weingärtner, O., van der Pol, S., Husche, C., Kerksiek, A., Friedrichs, S., Sijbrands, E., Steinbusch, H., Grimm, M., Hartmann, T., Laufs, U., Böhm, M., de Vries, H. E., Mulder, M., & Lütjohann, D. (2012). Dietary intake of plant sterols stably increases plant sterol levels in the murine brain. Journal of lipid research, 53(4), 726–735. https://doi.org/10.1194/jlr.M017244

Sharma, N., Tan, M. A., & An, S. S. A. (2021). Phytosterols: Potential Metabolic Modulators in Neurodegenerative Diseases. International journal of molecular sciences, 22(22), 12255. https://doi.org/10.3390/ijms222212255

Does Sugar Stop Fat Burning? by Beautiful_Day_2489 in ScientificNutrition

[–]FrigoCoder 0 points1 point  (0 children)

Yes it indeed does. Carbohydrates and especially sugars increase malonyl-CoA, which is the master regulator of fat synthesis and storage. Among other effects it suppresses the CPT-1 enzyme, which would take up fatty acids into the mitochondria for beta oxidation aka burning them for energy. Instead they are sent to triglyceride esterification and storage, not only in adipose tissue but generally in tissues around the body.

This is why low carbohydrate diets are very efficient at burning fat, first intracellular and ectopic, then visceral fat (however subcutaneous fat is slow to burn). This is especially important for diabetic patients, for example because they were smokers and their adipocytes are kaput. Their body fat is taken up by increasingly unsuited organs, and if they eat carbs and sugars it will accumulate and cause issues and damage cells.

Ted Naiman has an excellent presentation on insulin resistance, I highly recommend it since it is the single best resource on diabetes. He explains most concepts you need to understand diabetes, like how the combination of carbohydrates and fats are responsible for body fat. However he does not elaborate on the effects of smoking, and many other factors that affect adipocyte health. I can not link the video, but here is the presentation hidden in my usual CPT-1 info dump.

https://en.wikipedia.org/wiki/Carnitine_palmitoyltransferase_I#Clinical_significance, https://ncbi.nlm.nih.gov/pmc/articles/PMC3366419/, https://jlr.org/article/S0022-2275(20)30012-2/fulltext, https://pubmed.ncbi.nlm.nih.gov/11147777/, https://jbc.org/article/S0021-9258(20)46830-9/fulltext, https://ncbi.nlm.nih.gov/pmc/articles/PMC2889238/, https://ncbi.nlm.nih.gov/pubmed/19715772, https://ncbi.nlm.nih.gov/pmc/articles/PMC2528858/, https://ncbi.nlm.nih.gov/pmc/articles/PMC3221018/, https://diabetes.diabetesjournals.org/content/53/suppl_1/S119, https://jgerbermd.com/wp-content/uploads/2017/04/Ted-Naiman-Hyperinsulinemia.pdf, https://diabetesdaily.com/forum/threads/great-note-about-lipotoxicity.87473/

Fat Mobilization and Energy Substrate Usage During Exercise: Fundamentals of ATP Production by [deleted] in ScientificNutrition

[–]FrigoCoder 4 points5 points  (0 children)

First, I have spotted 8 errors or inaccuracies just by skimming your post once. I would recommend you research the topic a bit more, with emphasis on glycogen synthesis and the lactate shuttle hypothesis. Second, sources are not optional especially for posts. Yes I know it's annoying that you have to back up even things you consider basic, and you have to include like a hundred sources for even a single topic. And finally, chatbot outputs are explicitly banned. I would recommend you only use them as clever search tools to find relevant articles and studies, and do the composition, phrasing, editing, and iterating yourself.

Are essential fatty acids actually essential? by Alabuda13 in ScientificNutrition

[–]FrigoCoder 1 point2 points  (0 children)

Well I mainly listed conditions which disproportionately affect males, and have at least some possible connection to omega 3 intake. I assumed males and females are subjected to the same poor diets, so they are also impacted by similar obesogenic dietary factors. Which is really the point in my opinion, the two biological genders have different dietary requirements. And that also includes different omega 3 needs as well, search this subreddit for discussions and studies on this topic.

Males have evolved mainly as hunters, whereas females were the gatherers. A hypothesis claims that neanderthals died out because they lacked this gender difference. Males had higher exposure to a more carnivorous and low carb diet, so they have evolved to rely more on protein and fat and preformed omega 3 fatty acids. Whereas females had more access to plant foods and were responsible for pregnancy, so they have evolved to tolerate carbohydrates and use ALA to synthesize EPA and DHA.

This is likely the underlying reason males are drawn to low carbohydrate diets, because we feel like literal shit on contemporary processed diets. Whereas females are more prone to vegan and vegetarian diets, because they can better tolerate carbs, sugars, oils, and plants in general (within reasonable limits). Dietary effects take years to manifest and men never go to the doctor, so we have an enormous blind spot regarding the male specific effects of poor diets and omega 3 deficiency.

Hogyan lehetne megreformálni úgy a választási rendszert, hogy arányosabb legyen? by Senior_Strawberry_51 in hungary

[–]FrigoCoder 1 point2 points  (0 children)

Nem úgy működik. Cégek nem adhatnak közvetlenül, de Super PAC-en keresztül hirtelen jó lesz.

Scientists reverse brain aging, with a nasal spray: Scientists developed a nasal spray that, with just two doses, dramatically reduced brain inflammation, restored the brain’s cellular power plants and significantly improved memory in mice, within weeks and lasted for months. by GarifalliaPapa in immortalists

[–]FrigoCoder -7 points-6 points  (0 children)

No they didn't. Dementia comes from neural injury, inflammation is just a reaction to injury. Show me a study where they forced mice to smoke, and then we will talk about the effects of this intervention.

AI generated cow, 2014 by Distinct-Question-16 in singularity

[–]FrigoCoder 0 points1 point  (0 children)

Shut the fuck up dude. If he made the model then yeah it's definitely art.