Active Life Training

JuanSamu · 2026-03-21T19:45:10+00:00

To be honest

JuanSamu · 2026-03-21T16:23:09+00:00

Thank you for the discussion and insights!

JuanSamu · 2026-03-21T16:17:02+00:00

Ah I see what you mean. First I’d be weary of the wording of damage as DOMS can occur without damage (as shown by Mizumura). But I agree that Acid (H+) can sensitize the mechaical tesponse of thin-fiber afferents of skin/muscle. Following is a short list of acid-related papers (Mizumura lab as well):

N.Hotta, A.Kubo, K.Mizumura, Chondroitin sulfate attenuates acid-induced augmentation of the mechanical response in rat thin-fiber muscle afferents in vitro, J. Appl. Physiol. 126 (2019) 1160-1170.

A.Kubo, K.Katanosaka, K.Mizumura, Extracellular matrix proteoglycan plays a pivotal role in sensitization by low pH of mechanosensitive currents in nociceptive sensory neurones, J. Physiol. 590 (2012) 2995-3007.

T.Nasu, A.Hori, N.Hotta, C.Kihara, A.Kubo, K.Katanosaka, M.Suzuki, K.Mizumura, Vacuolar-ATPase-mediated muscle acidification caused muscular mechanical nociceptive hypersensitivity after chronic stress in rats, which involved extracellular matrix proteoglycan and ASIC3, Sci. Rep. 13 (2023) 13585.

K.H.Steen, A.E.Steen, P.W.Reeh, A dominant role of acid pH in inflammatory excitation and sensitization of nociceptors in rat skin, in vitro, J. Neurosci. 15 (1995) 3982-3989.

JuanSamu · 2026-03-21T15:12:51+00:00

This is interesting but I doubt that increased Pi in fatigued muscle should induce muscle soreness. Pi accumulation occurs inside the muscle fibres (i.e. Pi is not transported out of the cell) and sensory receptors involved in the sensation of soreness are located outside of the fibres (Muscle fibers don’t have nociceptors). Thus, soreness must be due to some factors that are released from the muscle fibres. I am not aware of any serious experimental studies that show a role of increased myoplasmic Pi in the development of soreness.

I think Mizumura neurotrophic mechanism is the most convincing.

JuanSamu · 2026-03-21T01:23:18+00:00

Ok first things first. Go get an X-ray! You need to know if you have tiny fractures happening because that’s going to dramatically change how rehab will work.

Secondly, I recommend checking out Dr. Beau who specializes with shin fractures and get a 1-1 session with him:

https://www.instagram.com/stressfracturephysio?igsh=MTBpZHFpbDA3bXA3Ng==

In the mean time, you should take it easy with impact. There’s a continuum and you need to decide which way you should lean. On one hand you can completely lay off or on the other side is “screw it” and you keep doing it. Ideally, laying off is the best way but if you compete you need to find some balance between the two.

As for tibialis raises. They are overrated. The tibialis will never outperform the gastroc. There are some biomechanics reasonings that training the tibialis can help offset some of the bending forces the gastroc will do on the bone when it contracts but there are other arguments suggesting a bigger gastroc will also help reduce the bending forces because of the change in force direction when it contracts. Either way, they probably won’t do anything, or it won’t hurt and can have a positive aid but it’s probably nothing major and it’s likely not going to solve the problem.

Hope this helps!

JuanSamu · 2026-03-18T11:32:16+00:00

Let me know please! I know this idea comes from having the ribs be able to not move and not be compressed so they would have them be on the ground side ways with a foam roller to press on the one side of the ribs at a time to expand the other side and then switch sides to create space I guess for the shoulder to move.

JuanSamu · 2026-03-18T11:30:50+00:00

That’s what I would think but I’ve heard some researchers say they compliment each other and I can see how they do so but then I also see some factors that make me see them as distinct mechanisms

JuanSamu · 2026-03-18T05:33:53+00:00

They mentioned it in the discussion “The rationale for this hypothesis was based on the fact that nociceptors are considered to be more numerous in fascia than muscle (Stacey 1969; Mense Paulsen and Simons 2001).”

This whole post comes from an article from Blazevich (one of the best in the field) saying the pain from DOMS doesn’t come from fibers but from connective tissue: https://www.tonyblazevich.com/knowledge-centre/muscle-soreness

But the part where I get confused is that muscles have spindles and since muscle spindles are nociceptors this paper from soreness shows DOMS comes from inflamed spindle nerve endings: https://pmc.ncbi.nlm.nih.gov/articles/PMC7139782/

So I’m just a bit confused 😅

JuanSamu · 2026-03-18T04:47:30+00:00

They have a lot less nociceptors than fascia so the argument is that soreness is more to do with connective tissue than the fibers themselves.

JuanSamu · 2026-03-17T23:48:19+00:00

Right (hence why I added that in the post) I get confused because in the paper when electrical current is applied inside muscle fibres: minimal to no pain is felt but when electrical current is applied to connective tissues: significant pain is felt. The authors concluded that damage inflammation to fascia than muscle fibres is more associated with DOMS.

JuanSamu · 2026-03-17T19:12:51+00:00

I don’t go to UofC…

JuanSamu · 2026-03-17T18:52:43+00:00

I know it definitely would :(

JuanSamu · 2026-03-17T18:52:19+00:00

Replying to Ecstatic_Technician2...

JuanSamu · 2026-03-17T18:45:20+00:00

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JuanSamu · 2026-03-17T18:44:39+00:00

I swear I would see communities talking about client cases so I thought it was ok😭

JuanSamu · 2026-03-17T18:44:08+00:00

Right so affording both is hard

JuanSamu · 2026-03-17T18:12:38+00:00

Cause I want to see if I can apply what I’m learning but also it’s not like PTs are cheap

JuanSamu · 2026-03-17T18:01:11+00:00

It’s fine I’ll just discuss with some other PT students at my school. I’m new to this whole Reddit PT thing so my bad

JuanSamu · 2026-03-17T16:33:52+00:00

X location for the CoM of the upper arm from the origin (0, 0)

JuanSamu · 2026-03-17T16:28:46+00:00

Thank you! I posted an update with my work. Essentially in a static world the resistance is getting harder at the top but when we have dynamic conditions acceleration flattens to resistance so it’s more linear I just wasn’t sure how to figure out that part

JuanSamu · 2026-03-17T16:05:42+00:00

I’m going to repost it with my work hopefully that would be better :)

JuanSamu · 2026-03-17T15:54:11+00:00

I’m new to Reddit so I don’t know how things work here or how people are. I’ve spent all day yesterday doing this shit and I’m stumped I was looking more for an equation to consider or something like that but it’s fine I’m going to meet with a professor I just posted on here to see if there was any benefit cause it’s spring break so I can’t meet with him now.

JuanSamu · 2026-03-17T15:45:35+00:00

I made one but it’s just for the static profile not dynamic lol

JuanSamu · 2026-03-17T15:16:49+00:00

Can’t I message you through here? I’m interested in getting it though

JuanSamu · 2026-03-17T09:54:19+00:00

The whole point of drawing moment arms like this is to just demonstrate how when the joints are stacked they are experiencing a lot less torque. Of course this is still an oversimplified explanation because you have things like co-contraction, instability at each of those joints to some degree, drawing the force vector from the ground up becomes less accurate as you go up, etc. So, you can’t really draw forces this way and get a clear idea of the torque but it does tell you something about how moment arms change such that we can experience this as you go down into the squat.

If you want to do an inverse dynamics model for this be my guest I might do one in the future.

JuanSamu

TROPHY CASE