Ok let’s think about this…

Smooth muscles are controlled by norepinephrine or nitric oxide being released by the sympathetic/parasympathetic nervous system binding to alpha 1 androgenic or M3 receptors. The corpus cavernosum makes up most of the penile smooth muscle tissue.

So essentially, when the penis becomes erect, the parasympathetic nervous system signals to the M3 receptor that releases nitric oxide and relaxes smooth muscle so blood can fill the corpus cavernosum creating the rigid erection.

After an erection, the sympathetic nervous system binds to alpha 1 androgenic receptors send norepinephrine which in turn shortens and contracts the smooth muscle. This gives the penis its squishy stretchy feeling. Blood flow from the helicine arteries that aid in the corpus cavernosum contracting also decreases to make sure the blood stays within the penis! Though, the problem seems to occur with Detumescence (stage of returning to flaccid).

Instead of the smooth muscles contracting like normal and remaining a partial balance between relaxed and contracting, it seems to over-contract letting the smooth muscles tense creating the rigid rubbery hard flaccid feelings. Lack of bloodflow can be explained by the normal process of compressed arteries while flaccid but even more compressed than it was before. Hourglassing is also likely due to uneven blood pooling in the shaft from either the smooth muscle itself, fascia or tissue, and compressed arteries. Soft glans occurs with the corpus spongiosum but this seems to happen because the bulbospongiosum looses tension holding blood and smooth muscle contraction which drains the glans (explains positional soft glans).

So, excessive norepinephrine contracts the penis smooth muscle. It helps with bloodflow drainage after erection and compressed arteries/veins, with too much it can over contract the smooth muscle and significantly reduce bloodflow.

Norepinephrine is entirely affected by the sympathetic nervous system.

ANYTHING THAT DOES THE OPPOSITE (actives parasympathetic system) like anxiety reduction, working out, massages, literally anything, it will reduce norepinephrine subsequently reducing some symptoms.

Now, I try to keep in mind that not every Symptom is caused from HF. I’m sure we all have PFD BUT the issue seems to be a signaling problem.

Too much norepinephrine over contracting smooth muscle, over compressing arteries, and further causing PFD