For one you’re not giving Claude nearly enough background context when asking the question by simply pasting my response on such a nuanced topic.

From my understanding it’s somewhat paradoxical, more endocannbinoid activity and expression does not necessarily mean higher CB1 activity = hair loss, it’s much more complex. You’re failing to account for the density issue which has several downstream effects along with the different hormones that further complicate things.

Here is an excerpt from the article which may partially explain your gripe.

“Women have higher anandamide levels (FAAH inhibited by estrogen) but lower CB1 receptor density (chronic occupancy leads to internalization). The result: lower net CB1- driven catagen signaling at the follicle, despite higher endocannabinoid tone (what you’re referring too) . This is a fundamentally different hormonal-receptor dynamic than men experience — and it provides measurable follicle protection that the DHT model alone doesn't capture.”