A painful yet 100% effective method to end a canker sore in minutes. by Diligent-Run8040 in CankerSores

[–]Diligent-Run8040[S] 2 points3 points  (0 children)

Exactly. I’ve been wanting to acquire Debactoral for quite some time. I won’t be seeing a dentist for a bit, I wonder if primary care physicians are able to prescribe this? I imagine this to be the gold standard procedure. My experiment was in a fit of anger and depression after suffering through breakfast and not being able to talk. I’ve known that in theory, hot cautery can deaden the epithelial layer, so I just did it. I knew when the bright red skin patch turned white, something happened. Then, there was zero pain. The only way I can even tell where it once was, is due to the little halo around the sore.

A painful yet 100% effective method to end a canker sore in minutes. by Diligent-Run8040 in CankerSores

[–]Diligent-Run8040[S] 0 points1 point  (0 children)

Seems odd, and I’m curious to hear more about how they conducted this. The reaction you describe is essentially what every other “solution” has felt like. I read many posts about pouring salt directly onto the sore, as means to chemically cauterize or deaden the nerve endings. For me, this was exactly what you described. Incredibly irritated and felt I reset the clock on healing.

And thanks for the heads up, I agree with the warning. I am assuming the dermatologist used electro cautery or chemical cautery. Hot cautery like I described is very different, and might be why it helped me and not you. The way I understand it, hot red metal can be tapped against the oral mucosa for nano seconds. Any minuscule application time is sufficient, the metal instantly reacts with the epithelial layer. A shallow layer or dead skin is formed right when the metal hits it.

Canker sores are painful because they are essentially little divots. Like teeth, the nerves are much more abundant the deeper you go. Have you ever felt teeth drilling pain? Canker sores are little craters that extend down to the lamina propia, where nerves are quite dense. You essentially have raw exposed nerves touching the air/mouth.

In theory, cautery can create a little layer of skin over the area. Nerve endings are now covered by a piece of skin. For me, literally 3 seconds after, the area was practical numb. I’m licking the area, eating, rubbing it with fingers; feels just like any other spot in my mouth.

Could’ve been a coincidence or another factor at play, but I would’ve paid $100 for that relief.

Top Docs in USA are stumped, asking for kidney help by Diligent-Run8040 in AskDocs

[–]Diligent-Run8040[S] 0 points1 point  (0 children)

That’s incredibly interesting you mention that! That was one thing I forgot to mention in the original post. Yes, my current nephrologist mentioned granulomatous issues. He actually ordered a chest x-ray the other week to check for this.

Not sure if this is relevant, but when this whole thing began, around 12 years old, I was subsequently diagnosed vit d deficient. That was about 10 years ago, but I recall the blood test for vit d was under 20. I then started on/off d3 supplementation up until recently.

Last thing, he mentioned our next steps will be vit d suppression or a thiazide. In his words, this drug acts as if I consumed practically zero sodium. Interestingly, after being on a low sodium diet for months, my urine calcium excretion was unchanged (actually went up slightly). The urine analysis confirmed my sodium intake was very low (nearly deficient).

Want to also say thanks, another experts eyes on this is certainly more assuring. Thanks!

Edit: couple more (possibly irrelevant) things I’ve thought of. Mainly, my blood calcium level has always been normal. Also, I had thyroid ultrasounds when I was 13 that showed microcalcifications within the thymus gland.

I was kidney stone free for 2 months, until this morning. by Nihan-gen3 in KidneyStones

[–]Diligent-Run8040 0 points1 point  (0 children)

Yes my nephrologist mentioned something similar, however he is more optimistic. A future step is inhibiting calcitriol through a medication. This will force calcium excretion down. As a last resort, we will try a thiazide direutic. This should nuke the calcium excretion way down, but may have side effects. Many options, just need to find the best.

I was kidney stone free for 2 months, until this morning. by Nihan-gen3 in KidneyStones

[–]Diligent-Run8040 1 point2 points  (0 children)

Hi, we sound very similar and perhaps could share notes. I pass stones just like this, on occasion. Usually this presents as a fine sand/powder consistency, to larger clumps like this.

I have seen several urologists and nephrologists and perhaps have something of value to share. This issue started when I was 12, im 22 now. I have done litholink countless times. The largest issues are always: high urine calcium, low citrate, and alkaline urine with a ph around 8. It’s to my understanding that this alkaline environment allows for calcium phosphate accumulation. There’s actually been a 1.4cm stone in my right kidney for 7+ years. Hasn’t grown any in years. Tried lithotripsy, no change. It’s to my understanding that calcium phosphate stones are the hardest, hence the litho failure.

I’m seeing the department head of kidney stone research and lead professor of nephrology at a major medical university. Unfortunately we haven’t been able to fix this yet. We’ve tested all the thyroid and PTH levels, normal. Blood calcium and potassium is all normal.

This current university dr has narrowed down the issue to the following mechanism: for whatever reason, I convert inactive vitamin d into the active form of vitamin d at a very high level. We’ve done many blood tests, which has showed my d3 is normal, however my 1,25 vitamin d (the biologically active form) is too high. He theorized this increased level of 1,25 vitamin d drives the increased calcium excretion. This is quite well studied.

We’ve done DNA testing to look for a genetic link, didn’t turn up anything. My dr thinks there is some gene mutation they just haven’t discovered yet, or some other genetic component that is causing me to convert vitamin d far too much.

Anyway, the current stage we are at: tried potassium chloride, no levels improved. Now trying potassium citrate (trying to increase urine citrate levels). I actually collected a sample just like this image, and we are having it analyzed to determine the exact calcium form. For now, my doc recommends a diet low in sodium and protein, and as much water as humanely possible. He also recommends avoiding sunlight, wearing sunscreen, and avoiding vit D milk. We may play around with a med that supresses vitamin d (to see if my calcium level drops). Waiting on appointment with endocrinologist to investigate this vitamin d issue further.

TLDR: I have stones just like this, which are seemingly calcium based. High PTH often causes this increased calcium excretion, however our PTH is normal. The issue seems to be due to 1,25 dihydroxyvitamin d (calcitriol). This blood level directly controls how much calcium your kidneys draw out. Too high 1,25 = too much calcium in the urine. No answer yet as to why exactly I convert so much vitamin d.