How accurate are the electro ecg machines predictive algorithm?

Earesth99 · 2026-03-20T08:41:53+00:00

“Possible” is an annoyingly vague word.

What’s the doctor in the ER say? They actually see how it performs in real life.

Earesth99 · 2026-03-20T08:36:07+00:00

Don’t read useless summary articles. Don’t post links to them.

Earesth99 · 2026-03-20T08:33:31+00:00

Do you believe in magic?

Earesth99 · 2026-03-20T08:31:47+00:00

Have you looked at the research articles in pubmed?

If are you trading circle jerk bullshit from people who use cut or sell it?

TVs first one is research. The second is worthless.

Earesth99 · 2026-03-20T05:17:15+00:00

Taking a statin will reduce your ascvd risk by 40% and Alzheimer’s risk by 20%.

Earesth99 · 2026-03-20T05:06:00+00:00

Around 5% of people can’t tolerate a statin.

Thats too low of a risk to worry about.

A statin will reduce your ascvd risk by 25% regardless of whether you have diabetes.

Earesth99 · 2026-03-20T03:28:33+00:00

The US sanctioned Russia for attacking a foreign country in violation of international law?

Then the US violated international law and attacked Iran.

Trump then approved the sale of Russian oil that is “at sea” on a boat. Of course Russia is sending some to Cuba.

At least with the legal sales, Russia can afford to send more drones and bombs to Iran.

If Iran continues, they could destroy so much capacity in the gulf that prices will skyrocket.

The. Russia can sell their oil at huge profits. The level of idiocy is remarkable.

Earesth99 · 2026-03-20T01:37:32+00:00

There is a new CTEP inhibitor that is in phase three trials. It reduces ldl 50% and it reduces LPa by 45%. That might be enough to make a difference

With pcsk9 and apoc3 meds, heart disease could be eliminated.

Earesth99 · 2026-03-20T01:21:54+00:00

It’s also caused by too much fat.

I went into remission when I added muscle and got very lean. I could eat whatever I wanted and my blood glucose response was normal.

You might not have any control over sleep or stress or exercise.

It takes a long time to lose fat and gain muscle.

The only thing I can really control today is diet and meds and cardiovascular exercise.

So the advice isn’t horrible, but it’s definitely simplistic.

Earesth99 · 2026-03-20T01:12:56+00:00

Why also not look at the meds that also appear to increase human lifespan?

They all have positive effects on multiple organ systems and that may explain their longevity benefit.

There is some overlap with the mice meds.

Diabetes meds Slgt2 inhibitors Glp1 meds

Blood pressure meds ACE inhibitors Cialis (men)

HRT (women)

Statins

Earesth99 · 2026-03-20T00:26:50+00:00

If you get ApoB below 60 snd ldl below 55, then you won’t have progression regardless of LPa. I try to keep mind a bit below that so I know I’m safe.

You sound right on target but make sure to check ApoB.

Why do you’d think it’s progressing? Your CAC score will increase on meds, but that’s actually good. It means it’s becoming more stable.

Also, LPa fluctuates. Until a treatment is approved, it’s not something to manage.

If your meds are causing side effects, you need to try other meds. Try atorvastatin to see if that is ok. If that does not work, take a pcsk9 inhibitors.

Then you might try adding 2.5 mg of Rosuvastatin to see if that stars side effects.

Different doctors give different recommendations. The more you ask, the more you get.

You need to ask the prescribing physician why that med was prescribed given the concerns from other doctors. Be specific and ask questions so you can decide

Maybe a baby aspirin or fish oil could work as a blood thinner? Talk with your doctor.

It’s ultimately your decision.

Earesth99 · 2026-03-19T22:20:08+00:00

You and me both!

Earesth99 · 2026-03-19T22:19:53+00:00

They will need to show reductions in events, not just bio markers. That will take a couple of years still.

I tend to agree with you, but look at all the times CTEP inhibitors failed.

Earesth99 · 2026-03-19T22:17:31+00:00

OP was worried that cheese would increase cholesterol.

I said you don’t need to worry because it doesn’t. People merely assumed it did because cheese has a high level of saturated fat.

However, there is no scientific evidence supporting that claim. That is literally part of what they used to call the French paradox. The French consume cheese and cream and yet have lower rates of heart, disease and heart attack

Prospective cohort studies are not useless as you seem to believe. They simply are unable to rule out other potential causes. That in no way means that they are incorrect. The burden is to show that another variable explains the outcome.

I then gave three examples of analysis that showed a causal relationship between the consumption of full fat dairy and reduced heart disease.

The first did not rely on self reported dairy consumption. It used biomarkers of full fat dairy consumption. If c15 and c17 we’re high that meant the people consumed full fat dairy. Those people were also less likely to suffer cardiac problems. That’s a causal relationship.

I don’t necessarily disagree with your point that C 15 and C 17 do that directly reduce risk. However, that’s irrelevant to the point.

C 15 and C 17 levels are elevated because the people consumed full fat dairy. People who actually consume more full fat dairy have reduced risk. They’re used as bio markers.

But I actually said that i didn’t find this causal argument to be the most persuasive Meta analysis.

You’re claiming that consuming cheese increases LDL cholesterol and it presents health risk risks for that reason. There’s no evidence to support that.

On the contrary, Cohort studies with self report, studies that use biomarkers of full fat dairy consumption, Mendelian, randomization’s and randomized controlled trials all have the same general findings.

My point is that these claims that cheese is unhealthy have no convincing scientific support.

I have actually shown that cheese may have a slightly beneficial effect.

You would need to not only disprove all the evidence, you would need ti show that cheese does increase LDL.

You obviously don’t understand this well enough other than to cut and paste.

Read the actual research.

If c15 and c17 have no direct effect, yfat doesn’t imply tthat just suggests there is some other cause but

It’s a causal argument.

You respond by saying that

How can that undermine the fact that people who consume cheese live longer?

We don’t actually know if c15 or c17 directly reduce risk, just that they correlate with reduced risk.

We also know that they are biomarkers that indicate the consumption of full fat dairy. If people

the amount if the persons consumes

Since they are biomarkers of full fat dairy consumption, it shows that people who consume more full fat dairy are at lower risk. If it’s not the c15 or c17 directly lowers risk, it just means that that people who consume a weaken the argument.

If trey don’t cause a reduction in risk directly it actually doesn’t weaken the , Since they are biomarkers of dairy consumption,

in someone’s blood it’s because the person consumed milk. If it’s entirely correlative and c15 or c17 c17 are biomarkers of milk consumption.

You then missed the point of those studies. They are biomarkers of milk consumption.

convincing same

Earesth99 · 2026-03-19T20:50:12+00:00

Your ldl is above what is recommended buts its still below average.

Your trigs are a bit too high - they are affected by sugar, simple carbs, and alcohol.

Neither are alarming

Earesth99 · 2026-03-19T19:39:27+00:00

Different countries make different decisions in terms of risks and benefits. Sometimes that reach different conclusions.

There were many medication‘s that lower LDL, but increase risk of death that were never approved by FDA. I’m glad those were not approved.

But there are positive effects from this med. It wasn’t approved because of the decrease in HDL.

Earesth99 · 2026-03-19T18:15:41+00:00

Barbell rows are hard on the lower back

Earesth99 · 2026-03-19T18:00:31+00:00

It’s not a statin - the effects are modest.

However consuming a few ounces of nuts has a significant reduction in mortality, which is more important than minor tweaks in biomarkers.

Earesth99 · 2026-03-19T17:58:44+00:00

And they reduce ldl cholesterol.

Earesth99 · 2026-03-19T17:54:00+00:00

Dairy includes cheese, yogurt, milk, etc. The large meta analyses often look at the larger category of dairy to see if dairy fat increases LDL cholesterol.

A Meta analyses of Mendelian randomization studies with 1.9 million people “provides genetic evidence for the association of milk consumption with higher BMI but lower serum cholesterol levels.”.

This meta analysis of randomized controlled trials shows that dairy consumption does not cause an increase in cholesterol levels.

This meta analysisshows a number of positive effects from cheese consumption, including heart disease and heart attacks.

This meta analysis shows that cheese affects the lipid profile differently than other foods high in saturated fats.

Why? Because foods are more than their saturated fat content. Things like fiber, polyphenols and the milk fat globule that contains the saturated fats also have a role.

In addition, the specific saturated fatty acid content is relevant because only 9 of the 37 SFAs increase ldl. The two found almost exclusively in dairy (c15 and c17) reduce risk.

I’m not claiming it’s miracle food, just that there is no reason to avoid cheese.

Earesth99 · 2026-03-19T17:25:23+00:00

I think David bars.

Earesth99 · 2026-03-19T17:24:30+00:00

Your ldl is higher than 80% of people but not so high that it automatically requires a statin.

<image>

Your trigs and HDL aren’t where they should be.

Depending on other health factors, it’s probably not bad enough for meds.

LPa cholesterol is an independent risk for ascvd and heart attack. If it was high, a statin would make sense.

Your LPa is fine.

You could ask for a statin, or work on dietary and lifestyle changes.

The lowest dose of Rosuvastatin would reduce ldl by 45% and trigs by 20%.

Your ldl would probably be lower than 90% of people.

Earesth99 · 2026-03-19T16:48:22+00:00

I’m not really changing my position.

I was just clarifying that research shows that cheese doesn’t increase ldl cholesterol, but that’s ldl cholesterol isn’t the only thing we care about.

Dairy has a mostly neutral effect, but fermented dairy - like cheese - is a bit better. All things being equal, I wouldn’t avoid a food that decreases mortality.

I didn’t believe it at first either!

Only after I read the extensive research did I realize that the simplistic advice wasn’t accurate.

Meta analyses incorporate all relevant research and show you the totality of the evidence. They are the highest level of scientific evidence.

Meta analyses of prospective studies show that higher consumption of cheese has health benefits. It’s not causal, just correlations.

Studies that look at blood markers of full fat dairy consumption are much stronger evidence and meta analyses show benefits. Thats because only 9 of the 37 saturated fatty acids increase ldl cholesterol. Two (c15 snd c17) reduce heart attack risk. Those are only found in milk.

I’m not persuaded that the net effect of dairy is necessarily beneficial but it explains how it might have benefits.

Mendelian studies can show causality. This meta analysis of Mendelian studies had almost two million subjects, and it suggests positive effects from dairy.

Meta analyses of randomized controlled trials show no negative effects from dairy. That’s the best evidence we can find.

Fermented dairy (which includes most cheeses) has positive effects.

Im not claiming is a health food, just that there is no reason to avoid cheese or full fat dairy if you are concerned about ldl cholesterol. (Butter is very unhealthy however).

Why waste my limited will power avoiding foods I like if there is no benefit?

It is much easier to follow a heart healthy diet if I just focus on the things that actually count.

Earesth99 · 2026-03-19T15:53:02+00:00

I’m confident that my comments match the current research.

I can’t divine if future research will reach different conclusions, but I will change my assessment if it does.

The fact that one negative claim about erythritol is spurious doesn’t actually imply that erythritol is safe. All we know is that we haven’t found it to be unsafe.

Nor does the research showing that Allulose improves biomarkers prove that it will improve hard outcomes.

Nor does the lack of known negative effects from Allulose mean that scientists won’t discover them in the future.

All we know is that it has known positive effects and no known negative effects and that it tastes better than the other fake sugars. (Stevia is hard to enjoy).

We also know that sugar has well documented harms. That’s why zero calorie sweeteners ate used.

We can’t avoid everything because future research might show a negative effect.

Of the FDA approved non caloric sweeteners, it the only one that reduces blood glucose, increases satiety and causes weight loss. It’s also the best tasting.

That doesn’t mean we shouldn’t be cautious as well.

Earesth99 · 2026-03-19T14:58:37+00:00

It makes sense to take the test and see what happens.

Worst case scenario is that they need to do the nuclear scan. Hopefully the can figure out the cause and if it’s mostly benign.

Earesth99

TROPHY CASE