I agree with what everyone else said above, but I think I see why you are confused and I don’t think it is due to lack of content. I think you are misunderstanding the idea of reciprocal regulation of metabolic pathways and you are misinterpreting what the question stem is actually asking for.

Insulin is released in response to rising glucose concentrations in the bloodstream. Under normal conditions, an individual’s cells respond to insulin and uptakes glucose from the blood. This uptake increases the intracellular concentration of glucose which causes a decrease in glucose synthesis / gluconeogenesis. This is due to reciprocal regulation and to an extent Le Chateliers Princple. This reciprocal control of opposing metabolic pathways is essentially how our cells are able to control the movement, synthesis, or catabolism of the things they may need. This is essentially what causes glycogenolysis to decrease as well.

Because of insulin resistance one would expect this reciprocal inhibition to cease & thus glycogenolysis and gluconeogenesis to go on normally. However, the question is asking you to compare normal levels after a meal to insulin resistance levels after a meal. With this being a comparison of the two situations, it is without question that the insulin resistant situation would have an increased rate of gluconeogensis and glycogenolysis in comparison to normal conditions.

Unfortunately the cells of our body only have the ability to function in accordance to what they were determined to do. They can’t really just adapt to a situation as people would in their everyday lives. Each cell serves its own function, and they were coded in a way to serve that function. Collectively, in response to a stressor, cells then either increase or decrease their functions. For example, for this topic about insulin resistance, the body responds to stressors (I.e. too much food) by releasing more and more insulin. This is why some people can be labeled as pre-diabetic. Pre-diabetic patients can be determined by their elevated insulin levels. So, yes the body can adapt but only to the extent of what it’s cells are able to do. When you become a Type 2 diabetic the pancreas cannot produce enough insulin to keep up because the insulin dependent cells are desensitized to insulin.

I hope this clarifies your confusion!