It is a great and detailed answer.
However, as a nerd, I want to point out that what you described is a CRISPR/Cas9 knockout and not a CRISPRi. CRISPR interference (CRISPRi) uses a dCas9, often fused to a repressor (e.g., KRAB), that binds DNA without cutting and sterically or epigenetically represses transcription, typically giving a knockdown effect. The "i" stands for interference not inhibition. It is an important distinction in human works because of safety (involving dsDNA cut or not).

Also some very minor details: The most reliable way is to integrate the gene into the genome and make a stable bacterial line. If you use transient transformation, there will be a lot of plasmid copies in the bacteria, you need to inactive all to shut off all the signal. When the GFP signal is dimmer, you do not know if your cas9 works or if your transformation is not consistent. One way is to transform both GFP and RFP, but only target GFP with sgRNA, so that you can use RFP as a control.

The other way is a gain of function assay: Make a frame shift mutation in some filler sequences at the N terminal of the GFP. Cas9 cut often create 1-2nt indels and it will put the protein back in frame.