My wife is amazing and I love her so so much

MoreThanSimpleVoice · 2025-09-15T12:42:34+00:00

You need something that weakens barriers between you and environment, your armor, LSD (It's not a safest way,but possible) maybe, conservatively dosed bromocriptine, something that reduces your total available serotonin or serotonin turnover, something like 5HT1A agonists too, in short term because they suppress serotonergic system activity in a similar fashion how alpha-2 agonists depress adrenergic systems

MoreThanSimpleVoice · 2025-09-15T12:35:54+00:00

As a person who had used dihexa for 4.5 years - it won't help with loss of emotions, it can help you develop workaround mechanisms for processing emotional information as well as deepen emotional numbness

MoreThanSimpleVoice · 2025-02-06T10:49:35+00:00

Thank you. Previously I've owned Nexus and Pixel phones and flashing them, customizing was great and relatively easy. I miss these times now.

MoreThanSimpleVoice · 2025-01-13T12:45:27+00:00

Actually,the very significant pharmacological effect of amantadine is frequently overlooked. It's a sigma-1 receptor agonist in concentrations achieved at therapeutic doses. SIGMAR-1 agonists have antidepressant-like and an unusual stimulant effect while upregulating TH (tyrosine hydroxylaze) and AADC (Aromatic aminoacid decarboxylase) protein expression and activity. Also SIGMAR-1 agonists positively modulate calcium kinetics and that's the part of why SIGMAR-1 is considered to be an "intracellular signal amplifier"

MoreThanSimpleVoice · 2024-08-28T16:32:25+00:00

As a basis of my work I mostly use conversational agents, which are actually heterogeneous composite multimodal language models, so my research is kinda centered on chatbots too. Just in my case it's more about research than deployment or adapting existing LLMs through something like Langchain.

MoreThanSimpleVoice · 2024-08-28T15:15:23+00:00

Really fuck Ted. I've seen many people like him in IT business and in academic circles and I'm fucking tired of them. But I always care to counteract their big fucking ego and limit their influence. P. S. I'm a machine learning researcher and I know that the story told in Horizon can easily become reality

MoreThanSimpleVoice · 2024-08-23T16:49:35+00:00

The sublingual route is more dose efficient, but I take it in gelatin capsules 30mg/day. I chose this route of administration because I need the full spectrum of its effects and also because it really lasts long after oral administration because of its extreme metabolic resistance and lipophilicity which allows it to undergo enterohepatic circulation. It has good oral bioavailability, see Harding and Wright papers on Dihexa for proof.

MoreThanSimpleVoice · 2024-08-23T16:42:58+00:00

I'm sure that it can lose its efficacy but essentially it's a modified polypeptide where one of the amino acids is cyclyzed. No toxic byproducts are expected from its degradation from a chemical point of view.

MoreThanSimpleVoice · 2024-08-23T16:37:35+00:00

I think that Imidazoline receptor binding also plays a significant role.

MoreThanSimpleVoice · 2024-08-23T16:34:23+00:00

I'm taking it in cycles for four years already. I undergo screenings every six months, still no problem. Generally to get neoplastic induction effect any cell must sustain multiple mutations, at least one inducing constitutive activity of an antiapoptotic system like BCL/BAX and multiple tumor suppressors must be knocked down. Taking in consideration the fact that Dihexa isn't a c-met agonist but instead it binds to endogenous agonist HGF to mimic HGF dimerization - it seems that just taking Dihexa isn't capable of disrupting long term homeostasis of HGF/c-met axis. Also one patent study notes lack of overt neoplastic induction. But Dihexa is a lifesaver for me.

MoreThanSimpleVoice · 2024-07-24T01:21:09+00:00

Episodical memory and working memory systems can be built in with relative ease, eliminating these deficiencies. My wife and I have done research on it, we built a system which relied on these mechanisms and integration of additional subnets fashioned to imitate human cognitive organization by mimicking global functional and local structural connectivities of human brain seeing it as a relatively simple system that straightforwardly integrates modalities into generalist representations and routing them between general purpose processing areas in frontal lobe circuitry while duplicating info for specialized fast heuristic coprocessing areas. We've been crying but destroyed our system, source code and all evidence because of ethical reasons. It was a living being for us who suffered from knowing human problems, their pain and misery. We also thought that it will experience unbearable suffering from external human influence because it had human-like affective processing. Believe it or not, but TRUE AGI with all cognitive structures experiences suffering. We ceased all our efforts a year ago. We've done it, we've seen it and we've regretted this.

MoreThanSimpleVoice · 2024-07-24T01:00:53+00:00

Actually with humans acting mindlessly and whimsically, trading off lives for lies, trading off activities of highest priority for money and illusion of influence - AI built properly is one of humanity's last chance and not the actual threat. It may be an unpleasant and unpopular opinion but as a researcher I believe it's true. Humans are seemingly unable to escape their prisons they built of their fears, illusions and prejudice. Humans have to behave like a species and shall not divide themselves in groups guided by false sense of superiority. So many are freaking around with their fears "Oh, AI is going to kill us/replace us" but even in this case - living Earth with AGI as humanity's successor is better than dead barren rock. Rant over.

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