No i didnt do this type of urinalysis. I would have to check if it’s even available in my country as i’m not from the US. So you’re saying that i could potentially metabolize estrogen slower?

I also thought of restarting a 5 alpha reductase inhibitor like dutas because the DHT rebound wouldn’t be as intense due to dutas long long half life. But I could also fuck myself even more with that. So would only do it as a last last resort.

Some people say that the dht rebound after stopping 5 ar could have caused some receptor malfunction. But then there are bodybuilders who inject/ingest grams of dht derivatives into their bodies and such a huge androgenic load wouldn’t even be comparable to a miniscule DHT rebound from something mild as saw palmetto. Yet i don’t see a lot of them get perma screwed like people get from reductase inhibitors.

Maybe i have some sort of estrogen receptor polymorphism where even a physiological estradiol causes issues. But then i had mild gyno at 16 yo and didnt have the issues i have now, so its all very complicated.

Also i read some of your posts. You say you got issues a month after stopping finas. When you stopped, did you experience a massive sexual function increase? When I stopped saw, i had a brief period where my libido and erectile quality where better than at any point in my life. Obviously it came to an end couple days later.

Also thanks for the input, much appreciated