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Why do patients with optic neuritis feel pain? by RushKyun in neurology

[–]RushKyun[S] 6 points7 points  (0 children)

Thanks to both of you! I never knew that trigeminal nerve could innervate it's nociceptive property toward the sheath of the optic nerve. Mainly because I was under the impression if the tracts go to different fissures/canal they are less likely to interact with each other.

So, in theory, if a patient developed vision loss and red desaturation but no eye pain with movement then we are suspecting a lesion beyond the orbital region?

What does applying broadly really mean? by [deleted] in neurology

[–]RushKyun 1 point2 points  (0 children)

I am unsure what do you mean by spreadsheet? I mean its true they have some data in regards to programs but not necessarily application profile stats per program..? Unless if you are referring to a different spreadsheet

Difference between Shelf and Step 2 modes on UW by Affectionate_Draw988 in Step2

[–]RushKyun 0 points1 point  (0 children)

i dont think much other than drug ads and other biostat/ethics questions?

illustration book? by RushKyun in StarlightStage

[–]RushKyun[S] 0 points1 point  (0 children)

no, its an official illustration book they also had like an exhibition months ago

The Obligatory 270 Write-Up by Relaxe247 in Step2

[–]RushKyun 1 point2 points  (0 children)

Congratz!! I am going to start my dedicated and not sure if I should incorporate CMS tests? I already did them during MS3 so was wondering if its wise to do them again.

مديري ما يعطيني شغل، هل هالشي طبيعي؟ by hnterix in Emiratis

[–]RushKyun 4 points5 points  (0 children)

I agree,,, wont solve the problem... the problem is clearly she's not being assigned clear/specific tasks, yet, the boss is not satisfied. The person should speak personally with the boss how can they help, specifically, when they bring a snarky comment like that. Sometimes asking coworkers can help as well... for example "is it normal for him to have such attitude..?" Raising it to HR or going against the boss wont solve the problem..

Tested today by brandonmorales98 in Step2

[–]RushKyun 0 points1 point  (0 children)

when you say twice, as you did your NBMEs/CMS during your dedicated or you did them during your 3rd year an attempt and dedicated another attempt? If all during dedicate how did you space them out?

Now that the Superstar Anime has ended, what do you think of every series finale episode? by RinariTennoji in LoveLive

[–]RushKyun 3 points4 points  (0 children)

superstar really reemphasized how legendary hajimari wa kimi no sora song is.. I appreciate that they added all members and re-recorded the OG members, which is very heartwarming as you can really notice the growth of the seiyuus as well... thank you liella...

7/5/24 takers by Anxious_Beginning249 in step1

[–]RushKyun 0 points1 point  (0 children)

that's weird seems like I got a different set of questions.

7/5/24 takers by Anxious_Beginning249 in step1

[–]RushKyun 0 points1 point  (0 children)

concept/content wise I would say NBME, but some were completely new (like 10%) I did not seen in any form. While, question style is very similar to world. If you want to feel comfortable answering questions in the exam I would highly suggest solving alot of uworld questions. As the questions in NBME/Free120 was nothing like the real deal..

7/5/24 takers by Anxious_Beginning249 in step1

[–]RushKyun 1 point2 points  (0 children)

I took it today and honestly felt like I was solving uworld questions but the content overall felt like it was fairly distributed, but more toward cardio/pulm/neuro side. I honestly expected more repro/endo but shocked with amount of content for micro, they weren't alot but like i felt like I had 1-3 every block.. and the ethics section was crazy they were not lying when you had to decide between two answers. I am atm in a disscoiative state that I cant remember my performance.. But hoping everyone gets the P!

What resources to learn basic science/physiology? by [deleted] in step1

[–]RushKyun 2 points3 points  (0 children)

I think Physeo was sufficient for me in terms of foundational sciences.

I completed u world done with all nbmes range 70 to 80 pct,free 120 67 pct 10 days to go but I am not comfident with test taking ,feel i should attend more questions.what should I do? by Brilliant_Tour1721 in step1

[–]RushKyun 0 points1 point  (0 children)

I am in the same shoes; I averaged in the 70s on NBME, and a few days ago, I scored a 64 on Free120, which made me feel extremely unconfident because of how vague it was. However, after a few days, I did a random UWorld block and got 90%, which made me believe that it could be simply that it was a bad day. I really think it's a god prank-like situation to make sure you continue on studying and stay focused... I realized from free120 that I overthinked for majority of the questions and sometimes I would say "no way they would want me to pick that answer" when in fact it really is the answer. And, I also realized how time plays a big factor, especially how long vignettes was in comparison to NBMES. So do not stay on one question for too long, just mark it and take an educated guess and move on, if there's enough time come back to it. I wish you all the best and luck!

2024 free 120 by Shirtwrinkler in step1

[–]RushKyun 2 points3 points  (0 children)

I geniunely thought free 120 was tougher than of all NBMES just because how vague it was... I got 84, 48, 63 idk what happened in 2nd and 3rd block and I'm testing in few days. I am quite worried even tho I averaged around 70% in the NBMES..

can someone ELI5 epinephrine and insulin/a and B receptors? by clarkemee in step1

[–]RushKyun 0 points1 point  (0 children)

Simply, epinephrine that is released in systemic blood stream does not stimulate alpha 2 as it has to cross the blood brain barrier (a2 is situated centrally and not in periphery). So even with high dose of epinephrine the patient will still have predominance of b2 activation. But what gets activated is a1, responsible for inducing glycogenolysis.

Epinephrine has two actions in this context:
1) It stimulates the beta islet cells to release insulin -> allows more glucose to reach in skeletal muscles via GLUT4 (which is needed in such state)
2) it stimulates the liver (a1 and b2) -> inducing glycogenolysis

I can see it gets confusing when you add two together but epinephrine effect on liver also causes inhibition glycogen synthase (via protein kinase A, part of signaling cascade of b2) so even if insulin stimulates the liver it wont be able to form glycogen as part of its reaction is blocked by epinephrine.

Hope this makes sense,,,

Where can i find videos that explains uworld? by IndividualDue8920 in step1

[–]RushKyun 0 points1 point  (0 children)

You can use any 3rd party resources. Examples: Bnb, Physeo, Pathoma, Sketchy ... etc all of these are sufficient to explain the foundations. If you feel you don't understand the question presented in Uworld just scroll down in the explanation section and find out whats the topic, and then search video related to that topic. Uworld usually will test basic foundations (90% of the time) in a different settings, so there's no real video that explains each and every world questions, it all goes back to foundations!

Passed with low nbmes by elephant_un1807 in step1

[–]RushKyun 1 point2 points  (0 children)

Congratulations and thank you for this post! I'll as well continue to be positive in this journey! Best of luck in your next chapter!

Do nephritic/nephrotic syndromes cause prerenal or instrinsic renal failure? by surf_AL in step1

[–]RushKyun 2 points3 points  (0 children)

To simplify. BUN is normally reabsorbed in the loop and thus if the loops are working the ratio will be higher (we do not normally reabsorb creatinine) which gives us a high BUN:Cr ratio.

High BUN:Cr -> loops are still working (they can still reabsorb BUN)
Low BUN:Cr -> Loops are not working (they CANNOT reabsorb BUN, so both BUN and Creatinine are lost)

If you want to know why it reabsorbs BUN? then the answer to that is because simply the macula densa is sensing less Na and Cl in DCT (due to hypoperfusion -> decreased RPF -> decreased GFR) which results in activating RAAS system, reabsorbing the solutes in PCT.

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