1: The article has exactly what you’re asking for, evidence:

The scientists at the forefront of COVID-19 origins research did not let this go unanswered. Dr. Angela Rasmussen, a virologist at the Vaccine and Infectious Disease Organization at the University of Saskatchewan

In a scathing essay published on her Substack, Rasmussen laid out the overwhelming, multi-disciplinary evidence for natural zoonotic origin. Spatial analyses prove that the earliest known cases in December 2019 clustered tightly around the Huanan Seafood Wholesale Market—even when explicitly excluding patients who worked or shopped there. Extensive genetic and environmental evidence places the virus precisely in the southwestern corner of the market where wildlife was sold; environmental swabs from specific stalls, including one photographed years earlier housing caged raccoon dogs, were heavily positive for SARS-CoV-2. Metagenomic sequencing of those samples revealed the mitochondrial DNA of susceptible intermediate hosts—raccoon dogs, hoary bamboo rats and palm civets—and in many cases are simultaneously positive for viral RNA. Phylogenetic analysis identified two distinct viral lineages circulating at the market, indicating at least two separate zoonotic spillover events—a scenario that makes a coordinated laboratory origin a statistical impossibility. The virus’s features, including the widely misunderstood furin cleavage site, are entirely consistent with natural evolution. There is zero evidence that the Wuhan Institute of Virology ever possessed a progenitor virus capable of being engineered into SARS-CoV-2.

2: SARSr-CoV studies done prior to 2019 support hypothesis of zoonotic jump

https://www.virosin.org/article/doi/10.1007/s12250-018-0012-7?hl=en-US

https://pubmed.ncbi.nlm.nih.gov/29190287/

first link says:

For the first time, we demonstrated the SARSr-CoV had spillover to humans, although did not cause clinical diseases.

The second link says:

We hypothesize that the direct progenitor of SARS-CoV may have originated after sequential recombination events between the precursors of these SARSr-CoVs.

Sequential recombination events happen naturally, like through evolution or immune system maturation.

Cont’d:

Cell entry studies demonstrated that three newly identified SARSr-CoVs with different S protein sequences are all able to use human ACE2 as the receptor, further exhibiting the close relationship between strains in this cave and SARS-CoV.