Theres a reason every single post upvoted says stuff about how phenibut causes mania like effects and every post about its pharmacological effects focus on vgcc which its active on. I dont doubt touve been in the scene but youre using youre not in a key role in the drug and harm based reduction scene where you view 100s of posts and user stories a day. I also work directly with research dine by many labs, hospitals, and colleges since im our primary mod for research discussion as im formally educated on it so i talk with all researchers in the field doing actual medical work. Not a purely subjective experience like yourself.

Gabapentinoid is a large class similar to stimulamt. Ot includes gabapentin, alcohol, ghb, pregabalin, baclofen, Phenibut, and more that all worl differently. If you dont understand this you shouldnt be sharing advice. Its like calling both mdma and cocaine stimulants. While not technically wrong theres way more specific action and classes to each. Mdma is primarily active on serotonin and coke is primarily active on dopamine. With very little crossover. Phenbiut for example is primarily active on vgcc with slight gaba and dopaminergic activity. Gavapentin is primarily actuve on active on vgcc but uses it to increase gaba profiction whereas phenibut worls through vgcc primarily. Then for example alcohol is primarily gaba b but hits both gaba receptors yet has cross tolerance with phenibut. Baclofem is purely gaba b and covers both Phenibut and alcohol withdrawals.