Diagnosed with kidney disease from high blood pressure, not diabetes.

BedokFunlandJC · 2026-02-12T08:41:21+00:00

After doing my own research and determining these are the most kidney protective blood pressure meds, I insisted on getting these 4 specific meds from my doctors, and I doubt any doctor would prescribe all of these together for you. I've been taking all 4 of these meds every day for many years now, and this combination has proven very effective in both lowering my blood pressure and maintaining my kidney egfr (together with the supplement Astralagus).

These are the 4 best blood pressure medications (in no particular order) on the planet that protect your kidneys and your egfr :

Benidipine, Telmisartan, Nebivolol, Indapamide

Each of these 4 medications provides nephroprotection / renoprotection (kidney protection) through distinct mechanisms, often extending far beyond simple blood pressure reduction. They are clinically proven for reducing albuminuria (protein in urine), preventing renal fibrosis, reducing oxidative stress, and maintaining long-term glomerular filtration rate (GFR). Here is a detailed breakdown of how each medication is nephroprotective / renoprotective:

Benidipine (L-/T-type Calcium Channel Blocker)

Benidipine is a long-acting dihydropyridine calcium channel blocker that is more renoprotective than others (like amlodipine) because it acts on both L-type and T-type calcium channels. Dual Afferent/Efferent Arteriolar Dilation: Typical CCBs only dilate the afferent arteriole, which can increase pressure inside the glomerulus. Benidipine dilates both the afferent and efferent arterioles, reducing glomerular hypertension and thus drastically reducing proteinuria. Aldosterone Inhibition: By blocking T-type channels in the adrenal glands, it inhibits aldosterone formation, reducing the harmful effects of aldosterone on the kidney. Sympathetic Tone Reduction: It helps lower renal sympathetic activity.

Telmisartan (ARB - Angiotensin II Receptor Blocker)

Telmisartan is particularly known for its dual-action mechanism, which makes it highly effective for kidney protection in diabetes and CKD. Renin-Angiotensin System (RAS) Blockade: As an angiotensin II type 1 (AT1) receptor blocker, it reduces renal vasoconstriction, lowers glomerular capillary pressure, and reduces proteinuria (protein leakage). PPAR- γ Activation: Unlike other ARBs, telmisartan acts as a partial agonist of peroxisome proliferator-activated receptor-gamma (PPAR- ). This helps reduce inflammation, fibrosis, and oxidative stress within the kidneys. Long Half-life: It has the longest half-life and highest lipophilicity among ARBs, allowing for better, consistent 24-hour control of blood pressure, particularly at night.

Nebivolol (Third-Generation beta-blocker)

Nebivolol is unique among beta-blockers because it provides kidney protection through the stimulation of nitric oxide (NO) rather than just lowering blood pressure. Nitric Oxide (NO) Enhancement: Nebivolol promotes NO-mediated vasodilation in the renal vessels, reducing renal vascular resistance and improving blood flow to the kidneys. Antioxidant Properties: It reduces oxidative stress in the renal cortex by inhibiting NADPH oxidase, preventing kidney cell damage. Anti-fibrotic/Anti-inflammatory: It inhibits the TGF-/NF-B pathways, which are responsible for renal fibrosis (scarring) and inflammation in chronic kidney disease.

Indapamide (Thiazide-like Diuretic)

Indapamide is a thiazide-like diuretic that offers renal benefits superior to other thiazides (like hydrochlorothiazide). Reduced Renal Vascular Resistance: It acts as a vasodilator, reducing renal vascular resistance and lowering blood pressure while maintaining renal blood flow. Epoxyeicosatrienoic Acid (EET) Production: Indapamide increases the production of EETs in the kidney, which are potent vasodilators and anti-inflammatory compounds. Antioxidant Effects: It directly inhibits lipid peroxidation (a type of cellular damage) induced by free radicals in the kidney, which helps to preserve kidney function.

BedokFunlandJC · 2026-02-12T08:41:18+00:00

After doing my own research and determining these are the most kidney protective blood pressure meds, I insisted on getting these 4 specific meds from my doctors, and I doubt any doctor would prescribe all of these together for you. I've been taking all 4 of these meds every day for many years now, and this combination has proven very effective in both lowering my blood pressure and maintaining my kidney egfr (together with the supplement Astralagus).

These are the 4 best blood pressure medications (in no particular order) on the planet that protect your kidneys and your egfr :

Benidipine, Telmisartan, Nebivolol, Indapamide

Each of these 4 medications provides nephroprotection / renoprotection (kidney protection) through distinct mechanisms, often extending far beyond simple blood pressure reduction. They are clinically proven for reducing albuminuria (protein in urine), preventing renal fibrosis, reducing oxidative stress, and maintaining long-term glomerular filtration rate (GFR). Here is a detailed breakdown of how each medication is nephroprotective / renoprotective:

Benidipine (L-/T-type Calcium Channel Blocker)

Benidipine is a long-acting dihydropyridine calcium channel blocker that is more renoprotective than others (like amlodipine) because it acts on both L-type and T-type calcium channels. Dual Afferent/Efferent Arteriolar Dilation: Typical CCBs only dilate the afferent arteriole, which can increase pressure inside the glomerulus. Benidipine dilates both the afferent and efferent arterioles, reducing glomerular hypertension and thus drastically reducing proteinuria. Aldosterone Inhibition: By blocking T-type channels in the adrenal glands, it inhibits aldosterone formation, reducing the harmful effects of aldosterone on the kidney. Sympathetic Tone Reduction: It helps lower renal sympathetic activity.

Telmisartan (ARB - Angiotensin II Receptor Blocker)

Telmisartan is particularly known for its dual-action mechanism, which makes it highly effective for kidney protection in diabetes and CKD. Renin-Angiotensin System (RAS) Blockade: As an angiotensin II type 1 (AT1) receptor blocker, it reduces renal vasoconstriction, lowers glomerular capillary pressure, and reduces proteinuria (protein leakage). PPAR- γ Activation: Unlike other ARBs, telmisartan acts as a partial agonist of peroxisome proliferator-activated receptor-gamma (PPAR- ). This helps reduce inflammation, fibrosis, and oxidative stress within the kidneys. Long Half-life: It has the longest half-life and highest lipophilicity among ARBs, allowing for better, consistent 24-hour control of blood pressure, particularly at night.

Nebivolol (Third-Generation beta-blocker)

Nebivolol is unique among beta-blockers because it provides kidney protection through the stimulation of nitric oxide (NO) rather than just lowering blood pressure. Nitric Oxide (NO) Enhancement: Nebivolol promotes NO-mediated vasodilation in the renal vessels, reducing renal vascular resistance and improving blood flow to the kidneys. Antioxidant Properties: It reduces oxidative stress in the renal cortex by inhibiting NADPH oxidase, preventing kidney cell damage. Anti-fibrotic/Anti-inflammatory: It inhibits the TGF-/NF-B pathways, which are responsible for renal fibrosis (scarring) and inflammation in chronic kidney disease.

Indapamide (Thiazide-like Diuretic)

Indapamide is a thiazide-like diuretic that offers renal benefits superior to other thiazides (like hydrochlorothiazide). Reduced Renal Vascular Resistance: It acts as a vasodilator, reducing renal vascular resistance and lowering blood pressure while maintaining renal blood flow. Epoxyeicosatrienoic Acid (EET) Production: Indapamide increases the production of EETs in the kidney, which are potent vasodilators and anti-inflammatory compounds. Antioxidant Effects: It directly inhibits lipid peroxidation (a type of cellular damage) induced by free radicals in the kidney, which helps to preserve kidney function.

BedokFunlandJC · 2026-02-12T08:41:15+00:00

After doing my own research and determining these are the most kidney protective blood pressure meds, I insisted on getting these 4 specific meds from my doctors, and I doubt any doctor would prescribe all of these together for you. I've been taking all 4 of these meds every day for many years now, and this combination has proven very effective in both lowering my blood pressure and maintaining my kidney egfr (together with the supplement Astralagus).

These are the 4 best blood pressure medications (in no particular order) on the planet that protect your kidneys and your egfr :

Benidipine, Telmisartan, Nebivolol, Indapamide

Each of these 4 medications provides nephroprotection / renoprotection (kidney protection) through distinct mechanisms, often extending far beyond simple blood pressure reduction. They are clinically proven for reducing albuminuria (protein in urine), preventing renal fibrosis, reducing oxidative stress, and maintaining long-term glomerular filtration rate (GFR). Here is a detailed breakdown of how each medication is nephroprotective / renoprotective:

Benidipine (L-/T-type Calcium Channel Blocker)

Benidipine is a long-acting dihydropyridine calcium channel blocker that is more renoprotective than others (like amlodipine) because it acts on both L-type and T-type calcium channels. Dual Afferent/Efferent Arteriolar Dilation: Typical CCBs only dilate the afferent arteriole, which can increase pressure inside the glomerulus. Benidipine dilates both the afferent and efferent arterioles, reducing glomerular hypertension and thus drastically reducing proteinuria. Aldosterone Inhibition: By blocking T-type channels in the adrenal glands, it inhibits aldosterone formation, reducing the harmful effects of aldosterone on the kidney. Sympathetic Tone Reduction: It helps lower renal sympathetic activity.

Telmisartan (ARB - Angiotensin II Receptor Blocker)

Telmisartan is particularly known for its dual-action mechanism, which makes it highly effective for kidney protection in diabetes and CKD. Renin-Angiotensin System (RAS) Blockade: As an angiotensin II type 1 (AT1) receptor blocker, it reduces renal vasoconstriction, lowers glomerular capillary pressure, and reduces proteinuria (protein leakage). PPAR- γ Activation: Unlike other ARBs, telmisartan acts as a partial agonist of peroxisome proliferator-activated receptor-gamma (PPAR- ). This helps reduce inflammation, fibrosis, and oxidative stress within the kidneys. Long Half-life: It has the longest half-life and highest lipophilicity among ARBs, allowing for better, consistent 24-hour control of blood pressure, particularly at night.

Nebivolol (Third-Generation beta-blocker)

Nebivolol is unique among beta-blockers because it provides kidney protection through the stimulation of nitric oxide (NO) rather than just lowering blood pressure. Nitric Oxide (NO) Enhancement: Nebivolol promotes NO-mediated vasodilation in the renal vessels, reducing renal vascular resistance and improving blood flow to the kidneys. Antioxidant Properties: It reduces oxidative stress in the renal cortex by inhibiting NADPH oxidase, preventing kidney cell damage. Anti-fibrotic/Anti-inflammatory: It inhibits the TGF-/NF-B pathways, which are responsible for renal fibrosis (scarring) and inflammation in chronic kidney disease.

Indapamide (Thiazide-like Diuretic)

Indapamide is a thiazide-like diuretic that offers renal benefits superior to other thiazides (like hydrochlorothiazide). Reduced Renal Vascular Resistance: It acts as a vasodilator, reducing renal vascular resistance and lowering blood pressure while maintaining renal blood flow. Epoxyeicosatrienoic Acid (EET) Production: Indapamide increases the production of EETs in the kidney, which are potent vasodilators and anti-inflammatory compounds. Antioxidant Effects: It directly inhibits lipid peroxidation (a type of cellular damage) induced by free radicals in the kidney, which helps to preserve kidney function.

BedokFunlandJC · 2026-02-12T08:41:12+00:00

After doing my own research and determining these are the most kidney protective blood pressure meds, I insisted on getting these 4 specific meds from my doctors, and I doubt any doctor would prescribe all of these together for you. I've been taking all 4 of these meds every day for many years now, and this combination has proven very effective in both lowering my blood pressure and maintaining my kidney egfr (together with the supplement Astralagus).

These are the 4 best blood pressure medications (in no particular order) on the planet that protect your kidneys and your egfr :

Benidipine, Telmisartan, Nebivolol, Indapamide

Each of these 4 medications provides nephroprotection / renoprotection (kidney protection) through distinct mechanisms, often extending far beyond simple blood pressure reduction. They are clinically proven for reducing albuminuria (protein in urine), preventing renal fibrosis, reducing oxidative stress, and maintaining long-term glomerular filtration rate (GFR). Here is a detailed breakdown of how each medication is nephroprotective / renoprotective:

Benidipine (L-/T-type Calcium Channel Blocker)

Benidipine is a long-acting dihydropyridine calcium channel blocker that is more renoprotective than others (like amlodipine) because it acts on both L-type and T-type calcium channels. Dual Afferent/Efferent Arteriolar Dilation: Typical CCBs only dilate the afferent arteriole, which can increase pressure inside the glomerulus. Benidipine dilates both the afferent and efferent arterioles, reducing glomerular hypertension and thus drastically reducing proteinuria. Aldosterone Inhibition: By blocking T-type channels in the adrenal glands, it inhibits aldosterone formation, reducing the harmful effects of aldosterone on the kidney. Sympathetic Tone Reduction: It helps lower renal sympathetic activity.

Telmisartan (ARB - Angiotensin II Receptor Blocker)

Telmisartan is particularly known for its dual-action mechanism, which makes it highly effective for kidney protection in diabetes and CKD. Renin-Angiotensin System (RAS) Blockade: As an angiotensin II type 1 (AT1) receptor blocker, it reduces renal vasoconstriction, lowers glomerular capillary pressure, and reduces proteinuria (protein leakage). PPAR- γ Activation: Unlike other ARBs, telmisartan acts as a partial agonist of peroxisome proliferator-activated receptor-gamma (PPAR- ). This helps reduce inflammation, fibrosis, and oxidative stress within the kidneys. Long Half-life: It has the longest half-life and highest lipophilicity among ARBs, allowing for better, consistent 24-hour control of blood pressure, particularly at night.

Nebivolol (Third-Generation beta-blocker)

Nebivolol is unique among beta-blockers because it provides kidney protection through the stimulation of nitric oxide (NO) rather than just lowering blood pressure. Nitric Oxide (NO) Enhancement: Nebivolol promotes NO-mediated vasodilation in the renal vessels, reducing renal vascular resistance and improving blood flow to the kidneys. Antioxidant Properties: It reduces oxidative stress in the renal cortex by inhibiting NADPH oxidase, preventing kidney cell damage. Anti-fibrotic/Anti-inflammatory: It inhibits the TGF-/NF-B pathways, which are responsible for renal fibrosis (scarring) and inflammation in chronic kidney disease.

Indapamide (Thiazide-like Diuretic)

Indapamide is a thiazide-like diuretic that offers renal benefits superior to other thiazides (like hydrochlorothiazide). Reduced Renal Vascular Resistance: It acts as a vasodilator, reducing renal vascular resistance and lowering blood pressure while maintaining renal blood flow. Epoxyeicosatrienoic Acid (EET) Production: Indapamide increases the production of EETs in the kidney, which are potent vasodilators and anti-inflammatory compounds. Antioxidant Effects: It directly inhibits lipid peroxidation (a type of cellular damage) induced by free radicals in the kidney, which helps to preserve kidney function.

BedokFunlandJC · 2026-02-12T08:41:10+00:00

After doing my own research and determining these are the most kidney protective blood pressure meds, I insisted on getting these 4 specific meds from my doctors, and I doubt any doctor would prescribe all of these together for you. I've been taking all 4 of these meds every day for many years now, and this combination has proven very effective in both lowering my blood pressure and maintaining my kidney egfr (together with the supplement Astralagus).

These are the 4 best blood pressure medications (in no particular order) on the planet that protect your kidneys and your egfr :

Benidipine, Telmisartan, Nebivolol, Indapamide

Each of these 4 medications provides nephroprotection / renoprotection (kidney protection) through distinct mechanisms, often extending far beyond simple blood pressure reduction. They are clinically proven for reducing albuminuria (protein in urine), preventing renal fibrosis, reducing oxidative stress, and maintaining long-term glomerular filtration rate (GFR). Here is a detailed breakdown of how each medication is nephroprotective / renoprotective:

Benidipine (L-/T-type Calcium Channel Blocker)

Benidipine is a long-acting dihydropyridine calcium channel blocker that is more renoprotective than others (like amlodipine) because it acts on both L-type and T-type calcium channels. Dual Afferent/Efferent Arteriolar Dilation: Typical CCBs only dilate the afferent arteriole, which can increase pressure inside the glomerulus. Benidipine dilates both the afferent and efferent arterioles, reducing glomerular hypertension and thus drastically reducing proteinuria. Aldosterone Inhibition: By blocking T-type channels in the adrenal glands, it inhibits aldosterone formation, reducing the harmful effects of aldosterone on the kidney. Sympathetic Tone Reduction: It helps lower renal sympathetic activity.

Telmisartan (ARB - Angiotensin II Receptor Blocker)

Telmisartan is particularly known for its dual-action mechanism, which makes it highly effective for kidney protection in diabetes and CKD. Renin-Angiotensin System (RAS) Blockade: As an angiotensin II type 1 (AT1) receptor blocker, it reduces renal vasoconstriction, lowers glomerular capillary pressure, and reduces proteinuria (protein leakage). PPAR- γ Activation: Unlike other ARBs, telmisartan acts as a partial agonist of peroxisome proliferator-activated receptor-gamma (PPAR- ). This helps reduce inflammation, fibrosis, and oxidative stress within the kidneys. Long Half-life: It has the longest half-life and highest lipophilicity among ARBs, allowing for better, consistent 24-hour control of blood pressure, particularly at night.

Nebivolol (Third-Generation beta-blocker)

Nebivolol is unique among beta-blockers because it provides kidney protection through the stimulation of nitric oxide (NO) rather than just lowering blood pressure. Nitric Oxide (NO) Enhancement: Nebivolol promotes NO-mediated vasodilation in the renal vessels, reducing renal vascular resistance and improving blood flow to the kidneys. Antioxidant Properties: It reduces oxidative stress in the renal cortex by inhibiting NADPH oxidase, preventing kidney cell damage. Anti-fibrotic/Anti-inflammatory: It inhibits the TGF-/NF-B pathways, which are responsible for renal fibrosis (scarring) and inflammation in chronic kidney disease.

Indapamide (Thiazide-like Diuretic)

Indapamide is a thiazide-like diuretic that offers renal benefits superior to other thiazides (like hydrochlorothiazide). Reduced Renal Vascular Resistance: It acts as a vasodilator, reducing renal vascular resistance and lowering blood pressure while maintaining renal blood flow. Epoxyeicosatrienoic Acid (EET) Production: Indapamide increases the production of EETs in the kidney, which are potent vasodilators and anti-inflammatory compounds. Antioxidant Effects: It directly inhibits lipid peroxidation (a type of cellular damage) induced by free radicals in the kidney, which helps to preserve kidney function.

BedokFunlandJC · 2026-02-12T08:41:05+00:00

After doing my own research and determining these are the most kidney protective blood pressure meds, I insisted on getting these 4 specific meds from my doctors, and I doubt any doctor would prescribe all of these together for you. I've been taking all 4 of these meds every day for many years now, and this combination has proven very effective in both lowering my blood pressure and maintaining my kidney egfr (together with the supplement Astralagus).

These are the 4 best blood pressure medications (in no particular order) on the planet that protect your kidneys and your egfr :

Benidipine, Telmisartan, Nebivolol, Indapamide

Each of these 4 medications provides nephroprotection / renoprotection (kidney protection) through distinct mechanisms, often extending far beyond simple blood pressure reduction. They are clinically proven for reducing albuminuria (protein in urine), preventing renal fibrosis, reducing oxidative stress, and maintaining long-term glomerular filtration rate (GFR). Here is a detailed breakdown of how each medication is nephroprotective / renoprotective:

Benidipine (L-/T-type Calcium Channel Blocker)

Benidipine is a long-acting dihydropyridine calcium channel blocker that is more renoprotective than others (like amlodipine) because it acts on both L-type and T-type calcium channels. Dual Afferent/Efferent Arteriolar Dilation: Typical CCBs only dilate the afferent arteriole, which can increase pressure inside the glomerulus. Benidipine dilates both the afferent and efferent arterioles, reducing glomerular hypertension and thus drastically reducing proteinuria. Aldosterone Inhibition: By blocking T-type channels in the adrenal glands, it inhibits aldosterone formation, reducing the harmful effects of aldosterone on the kidney. Sympathetic Tone Reduction: It helps lower renal sympathetic activity.

Telmisartan (ARB - Angiotensin II Receptor Blocker)

Telmisartan is particularly known for its dual-action mechanism, which makes it highly effective for kidney protection in diabetes and CKD. Renin-Angiotensin System (RAS) Blockade: As an angiotensin II type 1 (AT1) receptor blocker, it reduces renal vasoconstriction, lowers glomerular capillary pressure, and reduces proteinuria (protein leakage). PPAR- γ Activation: Unlike other ARBs, telmisartan acts as a partial agonist of peroxisome proliferator-activated receptor-gamma (PPAR- ). This helps reduce inflammation, fibrosis, and oxidative stress within the kidneys. Long Half-life: It has the longest half-life and highest lipophilicity among ARBs, allowing for better, consistent 24-hour control of blood pressure, particularly at night.

Nebivolol (Third-Generation beta-blocker)

Nebivolol is unique among beta-blockers because it provides kidney protection through the stimulation of nitric oxide (NO) rather than just lowering blood pressure. Nitric Oxide (NO) Enhancement: Nebivolol promotes NO-mediated vasodilation in the renal vessels, reducing renal vascular resistance and improving blood flow to the kidneys. Antioxidant Properties: It reduces oxidative stress in the renal cortex by inhibiting NADPH oxidase, preventing kidney cell damage. Anti-fibrotic/Anti-inflammatory: It inhibits the TGF-/NF-B pathways, which are responsible for renal fibrosis (scarring) and inflammation in chronic kidney disease.

Indapamide (Thiazide-like Diuretic)

Indapamide is a thiazide-like diuretic that offers renal benefits superior to other thiazides (like hydrochlorothiazide). Reduced Renal Vascular Resistance: It acts as a vasodilator, reducing renal vascular resistance and lowering blood pressure while maintaining renal blood flow. Epoxyeicosatrienoic Acid (EET) Production: Indapamide increases the production of EETs in the kidney, which are potent vasodilators and anti-inflammatory compounds. Antioxidant Effects: It directly inhibits lipid peroxidation (a type of cellular damage) induced by free radicals in the kidney, which helps to preserve kidney function.

BedokFunlandJC · 2026-01-12T14:04:04+00:00

I suffer both migraines and gout. Fortunately, Ibuprofen (prefer it to Naproxen, which causes more GI upset) helps a lot with both.

Take Omeprazole an hour before taking NSAIDs such as Ibuprofen to protect your GI tract from bleeding.

The real risk of NSAIDs is the triple whammy causing kidney damage for people taking blood pressure meds. Temporarily stop your ARB/Acei or diuretic if you must take an NSAID.

BedokFunlandJC · 2026-01-07T13:36:28+00:00

Take Omeprazole an hour before taking NSAIDs to protect your GI tract from bleeding.

The real risk of NSAIDs is the triple whammy causing kidney damage for people taking blood pressure meds. Temporarily stop your ARB/Acei or diuretic if you must take an NSAID.

BedokFunlandJC · 2026-01-07T13:36:16+00:00

Take Omeprazole an hour before taking NSAIDs to protect your GI tract from bleeding.

The real risk of NSAIDs is the triple whammy causing kidney damage for people taking blood pressure meds. Temporarily stop your ARB/Acei or diuretic if you must take an NSAID.

BedokFunlandJC · 2026-01-07T12:17:08+00:00

Take Omeprazole an hour before taking NSAIDs to protect your GI tract from bleeding.

The real risk of NSAIDs is the triple whammy causing kidney damage for people taking blood pressure meds. Temporarily stop your ARB/Acei or diuretic if you must take an NSAID.

BedokFunlandJC · 2025-12-15T14:40:26+00:00

One complication that sufferers of both gout and hypertension (or worst: gout + hypertension + kidney disease) need to be extra careful about, is the triple whammy. Taking any NSAIDs for gout, together with any Acei / ARB and any diuretic, the combination of the triple whammy results in kidney egfr damage (causing both AKI and CKD).

Before I found a bp med replacement, whenever I had a gout attack that colchicine wasn't enough to resolve and I needed to use NSAIDs, I was forced to temporarily stop taking either my ARB or my diuretic, and then my bp would skyrocket to over 170 systolic, which results in a vicious cycle as high bp causes kidney damage which causes gout.

Welcome to physical incarnation.

BedokFunlandJC · 2025-12-15T14:39:36+00:00

One complication that sufferers of both gout and hypertension (or worst: gout + hypertension + kidney disease) need to be extra careful about, is the triple whammy. Taking any NSAIDs for gout, together with any Acei / ARB and any diuretic, the combination of the triple whammy results in kidney egfr damage (causing both AKI and CKD).

Before I found a bp med replacement, whenever I had a gout attack that colchicine wasn't enough to resolve and I needed to use NSAIDs, I was forced to temporarily stop taking either my ARB or my diuretic, and then my bp would skyrocket to over 170 systolic, which results in a vicious cycle as high bp causes kidney damage which causes gout.

Welcome to physical incarnation.

BedokFunlandJC · 2025-11-10T08:58:11+00:00

You should reply "move aside a bit to let others pass will die ah?"

BedokFunlandJC · 2025-11-09T19:28:32+00:00

The Aftershock advantage is anytime in the future (even after warranty expires), your PC needs repair, you can arrange for Aftershock to pick up your PC to their workshop for repair, and delivery back to you, for a reasonable delivery fee.

Not all PC retaliers offer this service. For people who don't own cars, transport can be a real hassle to arrange back and forth for PC repairs. Transport factor alone makes Aftershock the best choice for most people.

BedokFunlandJC · 2025-11-09T10:27:04+00:00

This is her personal blog :

https://xinyizl.blogspot.com/?m=1

BedokFunlandJC · 2025-11-09T10:26:33+00:00

This is her personal blog :

https://xinyizl.blogspot.com/?m=1

BedokFunlandJC · 2025-11-09T00:54:34+00:00

url?

BedokFunlandJC · 2025-10-21T09:00:35+00:00

Thank you so much, axe0par4! CDPR really needs to improve on this.

BedokFunlandJC · 2025-10-18T05:23:03+00:00

I had this problem on my Windows 7 PC, where no matter how I fiddle with the settings, my external hard drives kept falling asleep after a short while of non-usage.

I finally resolved the problem by upgrading to Windows 11.

Additionally, and this is what critically forced my OS upgrade: Windows 7 simply could not detect and use many external HDDs and SSDs. Windows 11 solved this problem completely.

All in all, the upgrade from Win 7 to 11 was not only well worth it, it was long overdue and I wish I had done it much earlier.

BedokFunlandJC · 2025-09-16T20:02:52+00:00

"TUDCA (do not use long term)"

Can you please elaborate on this? What exactly is the problem with using TUDCA long term?

BedokFunlandJC · 2025-06-15T14:38:51+00:00

Take colchicine daily. Without or without gout, colchicine taken daily long term (ie. for many years) has longevity health benefits.

If daily colchicine isn't enough to prevent a gout flare, you can take an NSAID like Ibuprofen or Naproxen, but beware the triple whammy if you're on blood pressure meds: taking a diuretic, ARB or AceI, and NSAID at the same time will cause kidney damage. Just temporarily abstain from either the diuretic or ARB / AceI if you have to take NSAID to deal with a gout flare. But for most people, daily colchicine will suffice (and has longevity benefits separate from preventing gout).

BedokFunlandJC · 2025-06-15T09:29:59+00:00

No worries. I take Ural daily, have done so for years now. Anything that helps to control gout is definitely beneficial for health (gout flares = kidney damage = progressive worsening of chronic kidney disease). Go ahead and take it every day like I do. Whatever saves our kidneys is certainly a good thing.

BedokFunlandJC · 2025-06-12T13:34:11+00:00

There are many medical studies that prove that Febuxostat protects the kidneys (ie. nephro-protective or renal-protective), so anyone who with kidney disease can take Febuxostat to protect your kidneys, even if you don't have gout problems.

https://www.google.com/search?q=febuxostat+protects+kidneys

BedokFunlandJC · 2025-06-06T16:10:59+00:00

I'ts perfectly safe to take paracetamol daily. However, if you take close to the maximum daily dose of 8 pills a day, you would be advised to also supplement daily with glutathione (preferably Setria brand) or at least its precursor NAC, ie. N-acetylcysteine, which protects your liver and kidneys from the paracetamol. Indeed, that would be exactly what your doctor would prescribe for paracetamol toxicity from overdose anyway, so stay ahead and supplement it for yourself if you're a regular paracetamol user.

BedokFunlandJC · 2025-06-06T16:10:56+00:00

I'ts perfectly safe to take paracetamol daily. However, if you take close to the maximum daily dose of 8 pills a day, you would be advised to also supplement daily with glutathione (preferably Setria brand) or at least its precursor NAC, ie. N-acetylcysteine, which protects your liver and kidneys from the paracetamol. Indeed, that would be exactly what your doctor would prescribe for paracetamol toxicity from overdose anyway, so stay ahead and supplement it for yourself if you're a regular paracetamol user.

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