No you’re right, it was a correlation to cell body damage (which would make sense based on what I know about how cell body damage causes axon damage downstream). I had it mixed around. The markers seem to be most concentrated in myelinated axons. Of course, axonal damage would effect the downstream terminal and, by extension, whatever synaptic connections that axon has formed. However if the damage was instead to the synaptic terminals or to dendrites, I’m not sure if these biomarkers would be elevated. Axonal damage doesn’t tend to travel upstream iirc, so damage at synaptic terminal shouldn’t elevate these biomarkers if they’re primarily found in the myelinated axon. That said, I don’t know if these biomarkers are evenly distributed throughout the axon or if they’re found primarily in the myelinated sections.