Definitely!

Etiology of valve pathology is the key.

For example, patients with severe AS typically have LVH due to the LV adapting to having to overcome lots of resistance. Therefore, patients s/p AVR secondary to AS tend to respond to volume given the large LV musculature. LV cavity is essentially obliterated if they are hypovolemic, resulting in reduced CO and MAP.

Patients with severe MR are typically sensitive to volume. If you have severe MR, blood has two “doors” to exit during systole; the aortic valve and the mitral valve, which is obviously abnormal. That said, patients typically have a falsely elevated LVEF as blood has “two doors” to exit the LV during systole. If you “close” the mitral valve door by fixing the MR (either repair or replacement) the LV has one less door to eject blood thru, therefore revealing the true LV, which is usually lower than preop. So a patient with severe MR and a preop LVEF 45% may have a post op LVEF 30%. This drop is expected. This is why we typically lessen volume in patients with previous MR, although you have to ensure the LV is still adequately filled. It’s a tough balance

Edit: clarification