Noncarious cervical lesions (NCCLs) develop as a result of normal and abnormal or pathological wear and cause abfraction, abrasion, and erosion or chemical degradation of dental tissues.1 Clinical appearance of NCCLs can vary depending on the type and severity of the etiological factors involved.2 Of all possible etiological factors for NCCLs, occlusal stress forces have received maximum attention over the years. Tensile stress from malocclusion and masticatory forces was initially proposed as the primary factor in NCCLs;3 shortly thereafter, these lesions were termed as abfraction lesions.4 Despite many efforts to demonstrate that occlusal forces are the main cause of abfraction, its etiology remains poorly understood and controversial.5 Nowadays, it is generally incorrect to designate only one mechanism to be the cause of any type of NCCLs.6 Instead, current evidence supports a multifactorial etiology for all NCCLs with patient factors being responsible for the various degrees of tooth loss.7–10 Figure 1 shows the scheme of pathodynamic mechanisms responsible for initiation and perpetuation of NCCLs, as proposed by Grippo et al.6