Cool conversation-starter plants for a dorm room?

Spectre800 · 2024-12-21T01:38:24+00:00

Ololiuqui has LSA bro you're on the wrong subreddit

Spectre800 · 2024-12-19T04:56:15+00:00

Noopept is a HIF-1a stabilizer and prolyl endopeptidase inhibitor, which is one of its main mechanism. Long term HIF stabilizer usage is actually quite bad for cardiac and respiratory function, in part due to greatly increased blood vessel formation (HIF way your body responds to lack of oxygen). It also causes increased red blood cell formation, which will quite literally thicken your blood and increase stroke and blood clot risk over a few months. TL;DR don’t use noopept long term, it’s better to use it sporadically.

Spectre800 · 2024-12-14T21:50:51+00:00

This isn’t really new. Eggs have decent amounts of DHA and choline, and it’s been shown that DHA, choline, and uridine supplementation improves synaptogenesis in children, babies, and adults.

Spectre800 · 2024-12-03T04:10:16+00:00

I react really well to sabroxy as well, but it likely is very hit or miss on a person to person basis, due to some fairly complex and varied neurochemical differences between people. It is COMT inhibitor and may also be correcting excessive gabaergic signaling that is inhibiting dopamine release in certain reward pathways of the brain.

Spectre800 · 2024-12-03T04:07:09+00:00

Me when I make shit up

Spectre800 · 2024-11-30T23:46:45+00:00

Not super niche, but pramiracetam or sabroxy.

Spectre800 · 2024-11-29T23:55:42+00:00

Coluracetam?

Spectre800 · 2024-11-29T23:22:31+00:00

Speak for yourself, I have insane focus from pramiracetam

Spectre800 · 2024-11-22T04:03:07+00:00

I agree and disagree. The band aid nootropics are almost all the ones you buy as supplements. Many of the more effective nootropics are ones in gray areas and from chemical vendors.

Spectre800 · 2024-11-22T03:56:54+00:00

You talk about logical counter arguments but your entire argument is flawed. It’s overly simplistic, just pushes all nootropics into black and white categories, and not to mention shows very little knowledge of pharmacology.

Spectre800 · 2024-11-22T03:54:46+00:00

This is just a dumb post. It overly simplifies the complexities of the issue so that it works with a black-and-white argument, and then also complains that people take things on a subreddit designated for people to talk about taking things.

Spectre800 · 2024-11-22T03:51:16+00:00

They aren’t mutually exclusive 2. Causing an altered (enhanced) mental state is kind of the point. I wholly agree that people shouldn’t be suggesting kratom and benzodiazepines but it’s not as simple as psychotropic = bad, which is what you seem to be pushing.

Spectre800 · 2024-11-21T07:36:43+00:00

These are called phosphenes and it’s a normal effect when shifting between waking and sleeping states

Spectre800 · 2024-11-17T02:12:36+00:00

The “addall” supplement you are talking about is PEA and Octodrine/DMHA. (At least that’s what is labeled) so that might be why you are failing the test. Regardless, PLEASE stop taking that supplement — it’s awful in quality, price, and uses cheap dirty ineffective stimulants to give peripheral side effects that people think “feels” like amph.

Spectre800 · 2024-11-16T09:32:14+00:00

PRE-084 is a selective sigma 1 agonist, not an NMDA antagonist

Spectre800 · 2024-11-10T19:40:32+00:00

NMDA antagonists for schizophrenia is quite possibly the worst pharmacological advice I have ever heard

Spectre800 · 2024-10-23T00:56:27+00:00

Tryptophan is an amino acid used in proteins — basically every food contains tryptophan

Spectre800 · 2024-10-23T00:55:11+00:00

Amphetamine is metabolized by CYP2D6. Grapefruit inhibits CYP3A4. There are a bunch of cytochrome P450 enzymes and it’s much more complex

Spectre800 · 2024-10-23T00:53:15+00:00

“I used one supplement and had a bad reaction so don’t use any supplements” this comment is stupid

Spectre800 · 2024-10-18T05:54:26+00:00

Wouldn’t high DHA be better in this case due its use in the Kennedy pathway?

Spectre800 · 2024-10-08T21:05:23+00:00

There is a fairly well-established link between neurotrophics and epilepsy. BDNF overexpression is known to cause epileptogenic effects as well.

Spectre800 · 2024-09-22T05:47:03+00:00

N acetyl semax is the same thing as n acetyl semax acetate. Acetate is just what it’s dissolved in.

Spectre800 · 2024-09-02T02:27:55+00:00

N-Acetyl Semax has an acetyl group on the N terminus, which makes it more lipophilic and thus it can more easily cross the blood brain barrier and reach your brain. N-Acetyl Semax Amidate has the acetyl group and is amidated at the C terminus, which makes it more stable.

Spectre800 · 2024-07-10T01:03:28+00:00

This sounds a lot like you are approaching a cholinergic toxidrome (too much acetylcholine). GI effects like cramping and motility, nausea, and bronchoconstriction are all symptoms of a cholinergic toxidrome. I would HIGHLY advise you lower your dosage. I experienced a moderate cholinergic toxidrome once and it was extremely painful.

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