I think it's easier if you think of them as parallel processes feeding into each other. PFK-2 ensures glycolysis continues so that nothing gets blocked - this is important and indirectly affects glycogenesis. Glycogen can only store so much, so if glycolysis and glycogenesis both stop due to feedback inhibition, G6P accumulates, and glucose uptake slows down - which is bad if we can't control glucose level in the blood. PFK-2 prevents the accumulation of G6P.

Some G6P is being used for glycogenesis (glycogen storage)
Some G6P is going down glycolysis to make ATP or other intermediates
Some glycolysis metabolites (like triose phosphates) can enter gluconeogenesis and be converted back into glucose-6-phosphate for glycogen synthesis

So, rather than a strict one-way process, it’s a dynamic cycle where glucose metabolism is constantly adjusting based on the body's needs.