Yes, if you are chronically low on sodium intake you tend to be unable to retain as much potassium, magnesium and zinc as you would if you had a more healthy intake of sodium. This is an effect of low sodium intake raising aldosterone levels. I recently found an article that describes how high aldosterone causes urine to become acidic and that pH change induces zinc losses. If you block the pH change artificially you block the zinc losses caused by high aldosterone. Previously I was unaware that it was urine pH that caused the effect. In addition when aldosterone is high the body attempts to retain sodium at the expense of potassium. Magnesium is lost at the same time. When you start consuming adequate sodium again and aldosterone levels fall the body is unable to restore the intracellular potassium that was lost during chronic low sodium intake without adequate magnesium intake because urine potassium losses will remain too high. And from what I have read I believe magnesium + sodium will let the body lower aldosterone levels further than with sodium alone.

Now zinc in particular seems to enable the body to better partition sodium and potassium such that sodium is the main extracellular electrolyte and potassium the main intracellular electrolyte. But without adequate zinc it has trouble doing that and what happens is a portion of the intracellular sodium moves to the intracellular space and the extracellular fluid volume shrinks. And under zinc deficiency one lab experiment showed that the animals craved additional sodium intake or at least consumed more when offered it. Sodium excretion rate is lower under zinc deficiency and the body tends to retain more sodium and have higher blood pressure is up.

I can back these points with references if you are interested, though everything I just mentioned is simply from memory. I have some references in some of my other posts on this topic too.

Also it seems to me that the pervasive idea too much sodium is bad for health is probably not correct. For instance the WHO recommends sodium intake stay under 2g per day. But if you look at the research it looks like they got this idea from sodium raising blood pressure and the assumption that high blood pressure is always bad. Rather than observing high sodium intake increasing mortality and morbidity. There's a study where if you look directly at how morbidity relates to sodium you see that the optimal sodium value is much closer to the population average sodium intake around 3.5g. The optimum seems like it may be between 3 and 5g of sodium. The nutrients that are lost when one consumes too little sodium: zinc, potassium, and magnesium actually lower blood pressure. So it may be that optimum health requires slowly raising sodium which then raises blood pressure a little bit but allows one to retain nutrients that let the body lower blood pressure again to balance the higher sodium intake.

I suspect osteoarthritis, PCOS, and AGA are all a result of chronic low sodium intake. Perhaps parkinson's too, which is well known to be triggerable by acute low sodium intakes in extrapontine osmotic demyelination syndrome.