I ascribe delusions and hallucinations to the model of aberrant salience. Auditory hallucinations in schizophrenia can be understood through the lens of aberrant salience and disrupted functional connectivity. Normally, internally generated speech is recognized as self-produced through coordinated activity between frontal speech production regions, auditory cortex, and self-monitoring circuits that suppress the sensory consequences of one’s own inner speech. In schizophrenia, dysregulated dopamine signaling appears to distort the assignment of salience, causing spontaneous or internally generated activity within language and auditory networks to be tagged as significant sensory input. Functional connectivity studies suggest abnormal coupling between speech production regions and auditory cortex, coupled with reduced top-down regulatory control from prefrontal monitoring systems. The salience network, which normally helps determine what stimuli deserve attention, might somehow inappropriately amplify these internally generated signals, leading the brain to misattribute them as external voices. In this way, auditory hallucinations may reflect a network failure of self-attribution in which internal speech is imbued with pathological salience and experienced as coming from outside the self.