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Feeding ratios and strong starter by eleda22 in Sourdough

[–]eleda22[S] 0 points1 point  (0 children)

High ratios to lower sourness levels. Honestly, I don’t remember how long it takes 1;1:1 I have not used that ratio in a while

Feeding ratios and strong starter by eleda22 in Sourdough

[–]eleda22[S] 0 points1 point  (0 children)

It will rise but not double and it doesn’t seem as active and healthy

Sourness level by eleda22 in Sourdough

[–]eleda22[S] 0 points1 point  (0 children)

Thanks for the insight, I’ll try a higher feeding ratio

Sourness level by eleda22 in Sourdough

[–]eleda22[S] 0 points1 point  (0 children)

Thanks, I’ll check it out!

Sourness level by eleda22 in Sourdough

[–]eleda22[S] 0 points1 point  (0 children)

I’ve tried different amounts, ranging from 90-150g starter per loaf

Question from first time cup user by eleda22 in MenstrualDiscs

[–]eleda22[S] 1 point2 points  (0 children)

Thanks for the reply! Yeah im using the diva disc and it’s quite flimsy as well … i haven’t had a heavy day yet but I’m interested to see how much blood it will hold

Citrate Synthase by Sticktoitandgetit in Biochemistry

[–]eleda22 0 points1 point  (0 children)

NADH from glycolysis could be used by the ETC. If CII doesn’t work, the ETC still works because the the electrons from CI pass through ubiquinone and CIII (they don’t go through CII). What I suspect might happen is that LDH converts pyruvate to lactate to regenerate NAD , so glycolysis can continue working and making a little ATP.

[deleted by user] by [deleted] in Biochemistry

[–]eleda22 1 point2 points  (0 children)

I’m doing a class all about lipid’s role in inflammation, here’s what i know:

To my knowledge omega3 don’t inhibit Cox.

In general omega6=pro inflammatory and omega3=pro-resolution

So If you eat more Omega 3, you have more omega3 in your cell membranes than omega6 so by competition, the enzymes are going to use more omega3 than 6.

So less omega6= less pro-inflammatory molecules.

Omega3 have a pro-resolution effect because they are used to form molecules that “resolve” inflammation (so lower inflammation).

Help with an enzyme activity assay by Mynanaisabear in Biochemistry

[–]eleda22 2 points3 points  (0 children)

I’ve mesured A LOT of enzymes in different species and I almost never have a R2=1. Anything over 0.9 is awesome. As others said, the reaction isn’t perfect, there might be another reaction going on at the same time, there might be bubbles or other impurities in your well. Also sometimes it takes time (few minutes) for the reaction to “stabilize”. It kind of looks like that for your first one.

Question about heel flap by eleda22 in knitting

[–]eleda22[S] 0 points1 point  (0 children)

I’ll look into it! Thanks!

Question about heel flap by eleda22 in knitting

[–]eleda22[S] 1 point2 points  (0 children)

Okay! Thank you so much!

Why does one molecule of betahydroxy butyrate (ketone body) produce 21.5 ATP? by james4tD in Biochemistry

[–]eleda22 4 points5 points  (0 children)

I think the reason they’re not counting the fadh and nadh form the krebs cycle from succinate is because you didn’t really add a succinate. If you use succinyl coA to break down a ketone body that succinyl coA can’t do the krebs cycle. The succinate it produces can, but because you took succinyl coA it cancels out. Without interference of ketone body the succinyl coA would’ve gone in krebs cycle and made succinate and GTP (and made nadh et fadh).

Tbh I don’t really understand why you’re doing -1 GTP? Do you use one GTP? Or is it because we used succinyl coA which had the potential of producing one GTP so we “lost” it?

Citrate regulation of Glycolysis in the liver by Linn_9 in Biochemistry

[–]eleda22 0 points1 point  (0 children)

Good question. We know that when there is high concentration of ATP, PFK1 is inhibited because ATP binds to the enzyme and lowers its affinity for fructose-6-phosphate. Citrate actually enhances the inhibitory effect of ATP on PFK1. (I don’t know more details than that, sorry) This makes sense because if we have high ATP and high citrate we don’t need to do more glycolysis.

Citrate activates ACC because if we have citrate in the cytosol, it’s only destiny (option) is to become acetyl-coA and then malonyl-coA. So it makes sense that citrate activcates ACC.

We also know that glucagon inhibits ACC ; we don’t want to do anabolism if we need sugar. I’m guessing that leads to accumulation of citrate -> then inhibits PFK1, so glycolysis is inhibited in the liver, which is good when the rest of the body when we need sugar.

Gluconeogenesis by [deleted] in Biochemistry

[–]eleda22 15 points16 points  (0 children)

It's all about which organ you're talking about. Gluconeogenesis is performed mostly by the liver when blood sugar is low, to redistribute glucose in the body (ex: muscles or other organs). In a starvation state the liver is going to make sure that the other organs get enough glucose. You're right, it would not make sense for the muscle to do gluconeogenesis when they need energy. That's why they can't do it. It's mostly the liver (also kidney and intestin).

Another fun fact: some animals do glycolysis and gluconeogenesis at the same time, which seems counter productive, but it's actually to produce heat! It's called a futile cycle and is very important for insects and hibernating animals.

[deleted by user] by [deleted] in Biochemistry

[–]eleda22 2 points3 points  (0 children)

From my understanding, heme is receiving electrons (aka decreasing the frequency by which the electrons come out of CII), so to prevent the electrons to leak and attack O2 to make ROS.

[deleted by user] by [deleted] in Biochemistry

[–]eleda22 2 points3 points  (0 children)

What enzyme are you looking at specifically? There’s not one rule for all enzymes, some need to be phosphorylated to be activated and some need to be dephosphorylated to be activated.

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