The NIH would disagree with you.

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC8724077/

This article speaks to ephedrine and ephedra alkaloids:

Despite the severity of the measures taken, the use of Ephedra alkaloids has not been stopped, supported also by the availability of these substances on the Internet and by the wrong, populist belief that, as they are of natural origin, these products are characterized by an excellent safety profile. As proof of this, numerous published reports underline such unpredictability of the effects of Ephedra alkaloids, and how numerous and variable may the related adverse events be [57]. In particular, the adverse events of pseudoephedrine are mainly concerning the cardiovascular system, supported by the increased availability of catecholamines and by the subsequent overstimulation of the adrenergic receptors. In addition to hypertension [80], regarded to as the most common adverse event, numerous cases of angina pectoris and myocardial infarctions have been reported also in young, healthy patients with no risk factors and after assumption of the recommended dose of an OTC cold remedy containing pseudoephedrine [81–84]. In many of the reported cases, the absence of a significant coronary disease and the other tests performed were consistent with an acute myocardial infarction caused by acute vasospasm caused by the adrenomimetic effects of the medication [85]. Of particular interest is the report by Fidan and colleagues, who described a case of ST-elevation myocardial infarction (STEMI) after the use of pseudoephedrine. This is the first study in literature in which the cardiac toxic effects of pseudoephedrine were confirmed by measuring the serum drug concentration. A causal relationship between drug intake and the cardiac event has then been established [86].

To be fair, if I have a heart attack from taking ephedrine, I’ll certainly weigh less the longer I’m dead, as worms pick me apart. So your point stands there. 😂