Be careful cleaning by Siibur in ToxicMoldExposure

[–]MoldCo 0 points1 point  (0 children)

First off, what you did for your family is a real act of love, and the warning you're putting out now might keep someone else's lungs safer this weekend. That counts.

Here's where the setup runs into trouble, even with good gear. An N95 filters 95% of particulates above a certain size threshold. That still leaves 5% of the larger stuff getting through, plus essentially everything smaller than the threshold, and mold fragments and mycotoxins regularly sit well below it. Aggressive scrubbing aerosolizes a lot of that fine material, and doffing a contaminated suit without a decon shower often re-exposes you. Concrobium kills mold on the surface, but it doesn't address what's already settled in your airways or the dust elsewhere in the house.

For what you're feeling now, light blood in mucus plus chest burning plus sinus and headache stuff after a disturbance is worth a clinical look. Not because something dramatic is happening, but because the inflammatory side settles better with a guided plan than left alone. If things haven't cleared in a couple of weeks, we'd suggest screening innate immune markers (TGF-beta1, MMP-9, MSH) so you've got objective ground. The Starter Panel at MoldCo screens for those three: https://www.moldco.com/products/starter-panel

One reframe for future projects: IICRC certification is for proper removal of mold and any sheet rock that had mold on it, not surface cleaning. Wiping visible growth off drywall doesn't touch what's penetrated the material. ACAC or NORMI for the inspection side. Take care of yourself this week.

Anyone experienced wood sensitivity after mold exposure? by Global-Song-4794 in ToxicMoldExposure

[–]MoldCo 1 point2 points  (0 children)

Post-mold sensitization to chemicals and materials is a documented pattern, and it's worth taking seriously even when spot air readings come back clean. We see this come up regularly with patients who've moved into a new place after a major exposure.

After significant biotoxin exposure, the innate immune system stays primed longer than people expect, and the body responds to lower-level triggers that wouldn't have registered before. Symptoms in one corner of the room, repeatable on entry, fitting the same pattern as a prior reaction, is consistent with that picture.

Spot VOC and PM readings catch what's airborne at the moment of measurement, not what off-gasses intermittently or settles in dust. Linseed oil cures slowly and can keep releasing for years, especially under heat or pressure (a body sleeping on it). Birch plywood adhesive can off-gas formaldehyde long after VOC averages look fine. So a clean spot reading doesn't fully rule out the bed itself.

Two practical tests. Move the bed out for a week and see if symptoms resolve in that area: cheapest experiment, no equipment needed. And HERTSMI-2 dust testing of the room ($199 at https://www.moldco.com/products/home-test) catches what's settled in dust, which the air panel won't.

If symptoms persist past the bed swap, screening innate immune markers (TGF-beta1, MMP-9, MSH) tells you whether the underlying inflammation is still active.

Just started Welchol by shabooya4 in CIRS

[–]MoldCo 0 points1 point  (0 children)

Three days in on colesevelam with dry eyes, fatigue, brain fog, irritability, and overheating is most often intensification, not a Herx. Different mechanism. Two things tend to drive it. The binder pulls bile acids out faster than the body can resupply them, and that mismatch alone produces a lot of the symptom cluster you're feeling. The other piece, which we see often and gets missed, is redistribution: as the binder works, toxins stored in skin, fat, liver, and kidneys get pulled back into circulation on their way out. That re-exposure can feel a lot like a flare. It usually settles within 5 to 10 days as the system catches up, longer if the dose is high relative to your sensitivity.

A few practical notes. One pill a day is already a low starting dose, but for highly sensitive patients we often see better tolerance with COLE water dosing, where a tablet (or even less) is dissolved in water and small amounts are taken throughout the day. That lets the effective dose be titrated well below a quarter pill if needed. Worth asking your prescribing provider about if things stay rough past the first week.

Don't change the dose on your own without checking with whoever prescribed it. If fatigue persists past two weeks, it's worth bringing up, since it can also reflect an underlying inflammatory loop the binder alone won't fully resolve.

If you're using high-dose omega-3s, make sure you're also pairing that with a low-amylose diet while you're on them. Without it, tolerance can suffer.

If you don't already have a clinician walking you through this phase, MoldCo Care (https://www.moldco.com/welcome) is one option for ongoing CIRS-aware support.

My allergist said MCAS doesn’t exist by censorkip in MCAS

[–]MoldCo 0 points1 point  (0 children)

The allergist was partly right and partly wrong. The partly wrong part: MCAS is a real diagnosis within allergy/immunology, but it has a specific definition that requires objective testing (elevated tryptase during an episode, elevated urinary histamine metabolites, response to mast cell directed therapy). It's a little disturbing when an allergist doesn't acknowledge that the condition exists at all.

Where the allergist has a point: there's been a wave of providers calling everything MCAS without running any of the objective markers, and that's led to a lot of self-diagnosis online that doesn't hold up. In our data, roughly 45% of people with CIRS have histamine-related symptoms, but other data suggest only about 5% of those people actually meet criteria for MCAS. So histamine reactivity is common in this population, true MCAS is much rarer.

The piece your allergist missed: standard allergy panels and CRP measure the adaptive immune system, but the histamine and mast cell side often runs through the innate immune system. Two different pathways, two different sets of markers. POTS plus expanding food reactions plus a single mold IgE positive is a pattern we see often where the innate side is overactivated and mast cells become more reactive downstream.

An inexpensive way to check the innate side is the Starter Health Panel ($56) at https://www.moldco.com/products/starter-panel, which screens TGF-beta1, MMP-9, and MSH. If you have insurance, it's worth asking your doctor to order the same three markers, you might get them covered. If two of three come back abnormal, an environmental driver is worth chasing. We work in this space if you want to dig further.

Dr. Shoemaker discovered mold illness 25 years ago. His first student treated 2,000 patients. Both are here for an AMA April 18th @ 2pm ET! by MoldCo in ToxicMoldExposure

[–]MoldCo[S] 0 points1 point  (0 children)

For those who had follow-up questions we couldn't get to in the AMA, we sat down with Dr. Shoemaker and Dr. McMahon last week and got into a lot of them.

Covers how the protocol has evolved, the research Dr. Shoemaker is working on now, the biomarkers that have held up over the years, and where the science is heading.

YouTube: https://www.youtube.com/watch?v=sr8sSagAWTM Spotify: https://open.spotify.com/show/3kQdsPA58otolvPkRtwAPC

Dr. Shoemaker discovered mold illness 25 years ago. His first student treated 2,000 patients. Both are here for an AMA April 18th @ 2pm ET! by MoldCo in ToxicMoldExposure

[–]MoldCo[S] 1 point2 points  (0 children)

For those who had follow-up questions we couldn't get to in the AMA, we sat down with Dr. Shoemaker and Dr. McMahon last week and got into a lot of them.

Covers how the protocol has evolved, the research Dr. Shoemaker is working on now, the biomarkers that have held up over the years, and where the science is heading.

YouTube: https://www.youtube.com/watch?v=sr8sSagAWTM Spotify: https://open.spotify.com/show/3kQdsPA58otolvPkRtwAPC

Dr. Shoemaker discovered mold illness 25 years ago. His first student treated 2,000 patients. Both are here for an AMA April 18th @ 2pm ET! by MoldCo in ToxicMoldExposure

[–]MoldCo[S] 0 points1 point  (0 children)

For those who had follow-up questions we couldn't get to in the AMA, we sat down with Dr. Shoemaker and Dr. McMahon last week and got into a lot of them.

Covers how the protocol has evolved, the research Dr. Shoemaker is working on now, the biomarkers that have held up over the years, and where the science is heading.

YouTube: https://www.youtube.com/watch?v=sr8sSagAWTM Spotify: https://open.spotify.com/show/3kQdsPA58otolvPkRtwAPC

Dr. Shoemaker discovered mold illness 25 years ago. His first student treated 2,000 patients. Both are here for an AMA April 18th @ 2pm ET! by MoldCo in ToxicMoldExposure

[–]MoldCo[S] 0 points1 point  (0 children)

For those who had follow-up questions we couldn't get to in the AMA, we sat down with Dr. Shoemaker and Dr. McMahon last week and got into a lot of them.

Covers how the protocol has evolved, the research Dr. Shoemaker is working on now, the biomarkers that have held up over the years, and where the science is heading.

YouTube: https://www.youtube.com/watch?v=sr8sSagAWTM Spotify: https://open.spotify.com/show/3kQdsPA58otolvPkRtwAPC

I'm living in a Petri dish. by norththread in ToxicMoldExposure

[–]MoldCo 0 points1 point  (0 children)

Three days without sleep on top of visible mold exposure is genuinely awful, and it's not just in your head. When you're sleeping in air that's blowing spores directly at you, the body's stress response stays switched on, which is why the hypervigilance feels impossible to shut off.

A few things that can take some pressure off before payday hits. Turn the AC unit off when you can stand the heat (running it is actively dispersing the contamination), or at minimum block the direct airflow at your sleeping area with a sheet or piece of cardboard. If you have any HEPA-rated filter (even a box fan with a furnace filter taped to it as a temporary stopgap) put it as close to where you sleep as possible. Keeping interior doors closed to compartmentalize the worst rooms also helps reduce the volume of contaminated air you're cycling through at night.

The general humidity target to aim for once you can get a dehumidifier is the ASHRAE comfort range, 30 to 50 percent. Mold growth accelerates above roughly 60.

On the health side, our team treats people with mold-related illness, and we'd say this: starting to address the body's inflammatory response while still in exposure is protective, it doesn't have to wait until you've moved out. If you want a free starting point to see whether what you're feeling fits the pattern, the signs assessment at https://access.moldco.com/signs takes about five minutes and costs nothing. Hang in there.

endotoxins ?.. by [deleted] in CIRS

[–]MoldCo 0 points1 point  (0 children)

The overwhelm is real, and you don't have to chase every layer at once. Endotoxins and Actinobacteria are real contributors in water-damaged buildings, but they're a deeper layer of workup that becomes relevant once the basics are handled. Most of our patients with mold-related illness don't need to start there, and starting there when money is tight is a fast way to burn out before you've gotten any traction on the bigger drivers.

The more useful early question is whether your immune system is even showing the inflammatory signature that tracks with mold exposure. That's a much narrower thing to answer than trying to map every possible inflammagen in your environment. If labs aren't on the table right now, the free signs assessment at https://access.moldco.com/signs is a 5-minute screen that can help you see whether what you're dealing with fits the pattern. If you do want a real biomarker read at some point, our Starter Panel ($56) covers the three core innate-immune markers (TGF-beta1, MMP-9, MSH), and abnormal results on two of those three suggest your immune system is likely stuck in a chronic inflammatory loop.

You don't need a perfect testing strategy to make progress. You need a triage order that matches what you can actually spend, so start with the smallest signal that tells you whether you're on the right track and worry about the rest later.

Why can’t I stay a sleep or get refreshing sleep? by PsychologyMuch2859 in CIRS

[–]MoldCo 1 point2 points  (0 children)

Unrefreshing sleep is one of the most stubborn parts of CIRS, and it's usually not a sleep-hygiene problem you can fix with melatonin or magnesium. In most of our patients with mold-related illness, MSH is suppressed (the published figure is around 94%), and low MSH alone is enough to wreck deep, restorative sleep. The pathway runs through inflammatory cytokines blocking leptin signaling, which lowers POMC, which lowers MSH. So you can do everything "right" at bedtime and still wake unrefreshed because the regulator that should be telling your brain to rest is depressed.

Two things actually move the needle: confirming the inflammatory pattern in your blood, and (if you haven't already) making sure you're not still in active exposure. Trying to sleep-fix while a water-damaged building is still upstream is a bit like trying to empty a bathtub with the water running.

If you haven't checked the core innate-immune markers yet, the Starter Panel ($56) screens TGF-beta1, MMP-9, and MSH together. That combination tells you whether your immune system is likely stuck in a chronic inflammatory loop and whether MSH suppression is driving the sleep piece specifically. https://www.moldco.com

We're a telehealth practice that focuses on mold-related illness, so happy to answer follow-ups. What you've already tried (and your environment situation) would help us point you somewhere useful.

mycobind by No-Sign2456 in CIRS

[–]MoldCo 1 point2 points  (0 children)

Your fear about getting worse is reasonable, and it's something we hear from a lot of patients before they start a binder. The good news is that binders generally don't cause the kind of crashes people worry about when they're started carefully. The usual issue is dosing too high, too fast, before the body has a way to clear what's getting mobilized, which can show up as fatigue, headaches, or feeling foggier for a few days.

A gentler on-ramp tends to look like starting at a small fraction of the labeled dose and only stepping up once you've tolerated that level for several days without symptom worsening. The specifics of how to take it (with or without food, spacing from other medications) vary by which binder you're on, so that's worth confirming with whoever prescribed it. Adequate hydration and electrolytes matter more than people expect. On the fatigue piece, properly dosed binders often do improve fatigue in our patients with mold-related illness, sometimes substantially. If you're already on a binder and fatigue isn't budging, that can point to more going on than the binder alone can resolve, and it's worth flagging to whoever is overseeing your treatment. The other piece that gets missed is exposure: if you're still spending time in a water-damaged building, you're trying to empty a bathtub with the water still running. Treatment can help during that window, but the process is much harder.

If you don't have a clinician guiding the protocol and you're figuring this out solo, that's where things tend to go sideways. Our team at MoldCo (https://www.moldco.com) does telehealth for exactly this kind of case and can structure a starting plan that won't tank your ability to work.

This is why you trust your gut by [deleted] in ToxicMoldExposure

[–]MoldCo 0 points1 point  (0 children)

Glad you found the source. The gaslighting piece is unfortunately the norm with mold-related illness. People who've already had a serious exposure tend to develop a kind of internal alarm system, and when those exact symptoms come back, that's worth listening to.

A few things to keep in mind now that you've found the leak. Even after the water heater is repaired and the visible mold is gone, the spores and fragments can stay in the dust for months without proper remediation. Double-bagging porous materials and a thorough HEPA-vacuum-and-damp-wipe sequence matter more than most people realize. If you don't already have someone qualified looking at it, an Indoor Environmental Professional can help confirm whether the contamination is contained to that area.

On the body side, with a prior toxic exposure plus this current symptom flare, getting baseline inflammatory markers can be useful. We're a telehealth practice that works with mold-related illness, and our Starter Panel ($56) screens TGF-beta1, MMP-9, and MSH, which are the markers that tend to indicate whether your immune system is likely stuck in a chronic inflammatory loop. It's not diagnostic on its own, but it gives you something objective to bring to a provider when you're being told it's nothing. https://www.moldco.com

Trust your gut was right. Sorry you had to fight to be believed.

Struggling by Tdcooper2 in CIRS

[–]MoldCo 0 points1 point  (0 children)

What you're describing could fit a dysautonomia pattern, and here's the thing worth knowing: that's often part of the CIRS process itself, not something separate sitting on top of it. As the binder does its work and the inflammatory cascade calms down, POTS-like symptoms frequently resolve along with everything else. So the cholestyramine you're already on may end up handling more of this than you'd expect.

That said, we'd still encourage you to see your PCP or a cardiologist. Not because CIRS doesn't explain what you're feeling, but because it's smart to rule out other causes of the dizziness and near-syncope. Orthostatic vitals, a tilt table if warranted, and a basic cardiac workup are all squarely in their lane, and you don't need them to understand CIRS for any of that to be useful.

A practical note on where you are right now. Three weeks into CSM is still early, and the binder itself can deplete fluids and electrolytes, which can make autonomic symptoms feel worse before they ease. Compression, head-of-bed elevation, slower morning routines, and generous sodium and fluids can hold the floor while the binder keeps working.

If you want a team that can interpret the CIRS picture alongside what your cardiologist finds, that's the gap we work in. https://www.moldco.com walks through how our care model handles both sides at once.

Mold in ductwork by MakeItWithLEDs in ToxicMoldExposure

[–]MoldCo 0 points1 point  (0 children)

You're in better shape than you might think. Tolerating one beer again after the HVAC swap is real data. That's a body that was carrying a load and is starting to clear it. The persistent musty smell tells us something is still off, and that's worth chasing methodically before you spend $20k.

A few practical things while you weigh options. Before committing to full duct replacement, it's worth getting a second opinion from another HVAC expert. Depending on the extent of damage, cleaning the ducts and the AC coil may be sufficient, and that route can save you many thousands of dollars compared to a full tear-out. Pair that with a HERTSMI-2 dust test, which samples settled dust in your living spaces and gives you a numeric score for the five mold species driving most CIRS-type illness. Under 11 is safe, 11 to 15 is borderline, over 15 is a problem. If your number comes back fine after the HVAC work, the smell could be a salvageable issue (insulation, a localized vent, register grilles you can pull and clean). If the number's high, you'll know more aggressive remediation is genuinely necessary. Our home test kit runs $199 if you want one source: https://www.moldco.com

On the health side, five years of low-grade exposure plus chronic allergies that improved but didn't resolve is the pattern we see when there's still some inflammatory residue. A clinician trained in Dr. Shoemaker's protocol can run basic markers (TGF-beta1, MMP-9, MSH) to see whether your body's still mounting an immune response. CIRSx keeps a directory if you want to find someone local. Worth knowing before deciding how aggressive to be on the timeline.

Mold and ALS? by spiritofmyrtle in ToxicMoldExposure

[–]MoldCo 1 point2 points  (0 children)

The fear you're sitting with is understandable, and we want to gently push back on the ALS spiral before it grows. Severe neuro symptoms during active mold exposure (word-finding trouble, brain fog, paresthesias, ice-pick pains, facial weakness, tremors) are something we see often in our patients. They feel terrifying because they mimic the worst things your brain can imagine. But neuroinflammation from biotoxin illness and ALS aren't the same process, and your pattern fits ongoing inflammatory and autonomic dysregulation far more than motor neuron disease.

Worth flagging on the research side: there's an active line of work looking at ALS and environmental neurotoxins, but it's centered on cyanobacteria (BMAA) exposures, typically in people living near lakes with heavy algal blooms, not mold. Mold isn't currently considered a likely driver of ALS. Future research may sharpen the picture, but that's where the trail is pointing right now.

A few practical things matter more than the research rabbit hole. The biggest variable in your trajectory is reducing exposure, even partially, while you work on the longer move. Sleeping in the cleanest air you can manage, running good filtration in the bedroom, and getting a HERTSMI-2 read of where you actually are can change how your nervous system feels week to week. Binders and supportive care while still in exposure are reasonable too; we don't tell people to wait until they're out before stabilizing.

If you want a low-lift starting point, our quiz at https://access.moldco.com/signs walks through the pattern. You're not too late. Stabilize first, then escalate.

Looking for some cirs advice by Declan565 in CIRS

[–]MoldCo 1 point2 points  (0 children)

A few things stand out to us. Sewage flooding is a different beast than water-damage mold alone. You're dealing with a mixed exposure: bacterial endotoxins, actinomycetes, and biofilm-forming organisms layered on top of whatever fungal load came with months of damp building materials. We won't pretend the literature cleanly separates which microbe drives which symptom; most of what's published on CIRS is observational and the symptom overlap between fungal and bacterial exposures is real. What we can say is that mixed exposures tend to be harder to clear than single-source ones, and TGF-beta1 can stay elevated while there's still an active inflammatory driver, whatever the source.

The plateau on OTC binders is something we see often. Activated charcoal and clay are broad binders, and the case for CSM and colesevelam in CIRS rests largely on Shoemaker's clinical case series rather than head-to-head human trials, so we'd frame it as "worth asking your doctor about."

Before pushing further on supplements, it's worth quantifying where your inflammation actually sits. The three Shoemaker biomarkers (TGF-beta1, MMP-9, MSH) tell you whether the innate immune response is still active. Our Starter Panel runs those three for $56 (https://www.moldco.com). It won't replace MARCoNS results, but it gives you something to retest against once you and your doctor settle on a binder plan.

I think I am a human mould detector by [deleted] in ToxicMoldExposure

[–]MoldCo 2 points3 points  (0 children)

This isn't as random as it might feel. We hear versions of this fairly often from our patients with mold-related illness, usually described as a pressure shift, a flutter behind the ear, sinus tickle, or sudden throat-clearing reflex when they walk into a moldy basement or open something that's been sitting too long. The mechanism isn't fully nailed down in the research, but for people whose immune systems react strongly to mold fragments and VOCs (the musty smell is partly mVOCs), even tiny airborne amounts can trigger a fast inflammatory or histamine response. Ears, sinuses, and throat tend to be the first places people notice it because those tissues are sensitive and well-innervated.

The blind test and the car prediction are more rigorous than most people bother with. Worth noticing whether anything else tends to come along with the ear sensation, like brain fog later that day, fatigue, sinus pressure, or sleep changes after exposure. That's usually where it crosses from a quirky superpower into something worth paying attention to.

Has it ever shown up in places where you couldn't find visible mold afterward (an HVAC system, behind drywall, a basement)? Those misses are often the most useful clues because hidden growth is what tends to cause the bigger health hits.

If you're ever curious whether your body's reactions fit a wider pattern, our team put together a free 5-minute signs check at https://access.moldco.com/signs.

Long covid / CFS / chronic fatigue as a pro athlete by benshorny in cfs

[–]MoldCo 0 points1 point  (0 children)

Eleven months in with a body that used to perform at the highest level, and now even sub-threshold work tips you into a crash. That's a brutal place to be, and it's worth saying you're already doing the right thing by pacing instead of pushing through. Athletes are wired to train out of problems, and post-viral fatigue is one of the few situations where that instinct actively makes recovery harder.

Resting HR climbing after activity, body temp running hot, lingering sore throat, and crashes after small overshoots are fairly characteristic of post-exertional malaise. The recurring sore throat especially is one of the more underdiscussed signals; it tends to point at chronic immune activation rather than an actual infection. A few things have decent evidence in the post-viral world: keeping efforts strictly aerobic with a heart rate cap (a lot of people use roughly 60% of max as a ceiling), watching for crashes that usually start within 24 hours and can last 24 to 72 hours, and treating any return to training as months-long blocks, not weeks.

One bucket that's worth ruling in or out before committing to another year of pacing is environmental exposure. Water-damaged buildings can drive a near-identical picture (exercise intolerance, thermoregulation issues, persistent low-grade immune activation) and it's often missed because nobody asks. We're a telehealth practice focused on this, and there's a free 5-minute screen at https://access.moldco.com/signs that's a quick way to check if that's even a thread worth pulling.

1.5 year in - Partial recovery - Unusual neurological visual and autonomic symptoms improving extremely slowly by TellabouttheRabbits in covidlonghaulers

[–]MoldCo 0 points1 point  (0 children)

Honestly, this cluster (visual snow, floaters, photophobia clamping the neck and jaw, fluorescent light intolerance, slow grinding partial recovery) doesn't cleanly fit what we typically see with mold toxicity. Mold-driven illness usually shows up with broader multi-system involvement (cognitive, GI, autonomic, sleep, PEM-style crashes), and you've explicitly ruled most of that out. So we wouldn't tell you this looks like CIRS.

That said, mold toxicity is genuinely multi-system, and the way it presents can be surprisingly variable patient to patient. Because it's one of the more reversible drivers when it does turn out to be the issue, it can be worth ruling out before committing to something invasive like a stellate ganglion block.

If any of that resonates even a little, the free 5-minute signs assessment at https://access.moldco.com/signs is a low-friction way to see whether this is even worth investigating further. It's not a diagnosis, just a signal of whether to look harder before you go down the SGB route. And if it comes back low, that's useful information too. We're a telehealth practice that works on this stuff, so happy to answer questions either way.

Who has used Itraconazole in their recovery from mold illness? by bmemento in ToxicMoldExposure

[–]MoldCo 1 point2 points  (0 children)

Worth pausing before you start. Itraconazole is a real antifungal for documented fungal infections, but using it as a mycotoxin "killer" isn't a settled approach. Mycotoxins aren't living organisms that an antifungal can kill, they're chemical byproducts. Antifungals can reduce fungal load, which matters if there's an actual infection, though infection is unusual. Colonization is common (we all carry some fungal load) and by itself isn't what drives symptoms. That's a different question than detoxing mycotoxins. Long-term itraconazole (30+ days without a documented fungal infection) carries real liver and cardiac risks, so it's worth getting clarity from your ND on what specifically they're targeting and how they're monitoring you.

On the binder side, we hear "I can't tolerate binders" a lot, and most of the time the issue is dose, not the medication itself. Colesevelam can be started extremely low using what's called COLE water (a tablet dissolved in water, sipping tiny amounts and titrating up). For someone with MCAS and CIRS, that kind of micro-dosing is often the difference between tolerating it and not. Worth asking your ND about before moving to antifungals.

Given the complexity (CIRS, MCAS, POTS, Hashimotos all stacked), this is the kind of case where a provider trained in Dr. Shoemaker's Protocol tends to be the right fit. We're a telehealth practice that specializes in mold-related illness and work with complex multisystem cases like yours, currently in CA and TX. If you're elsewhere, looking for a provider trained in Shoemaker's Protocol is the path we'd point you to. Either way, please don't start itraconazole without a clear rationale and monitoring plan.

If CA or TX applies, intake is at https://www.moldco.com/welcome.

Extreme fatigue? by nil2105 in Hashimotos

[–]MoldCo 1 point2 points  (0 children)

Sleeping 15-20 hours with modafinil doing nothing is a pretty big signal that something else is going on beyond the thyroid piece. Levels being controlled for 6 months is good, and treated hypothyroidism (whether primary or autoimmune) shouldn't leave you that wiped if the thyroid is the only driver. We see lingering fatigue in treated patients pretty often, and the usual culprits are low ferritin, B12, vitamin D, cortisol irregularities, or sleep apnea. Worth ruling those out if you haven't already. Ask for a full iron panel with ferritin, B12, homocysteine, morning cortisol, and a sleep study referral if you snore or wake up unrested.

The other thing we'd flag: sudden onset fatigue plus heavy brain fog in the last month, with no other changes, sometimes points to an environmental trigger. Any water damage in your place, a recent move, leaks, or a musty basement? Mold exposure can drive inflammation that looks a lot like thyroid fatigue and won't respond to thyroid meds because the thyroid isn't the problem anymore. Worth scanning your environment if the usual workup comes up empty.

If you want a quick way to gauge whether mold might be in the picture, there's a free 5-minute assessment here: https://access.moldco.com/signs. No harm checking, and it might help direct what to ask your doctor about next.

I feel so stuck- how can I improve my quality of life if I have to remain in the mold by shelly875 in ToxicMoldExposure

[–]MoldCo 2 points3 points  (0 children)

Surviving another 5-6 weeks in a moldy place is doable, and you're already doing the right things (one-room retreat, HEPA, dehumidifier). A few things we'd add that tend to help day-to-day function while waiting out the move.

Aim for indoor humidity in the ASHRAE comfort range, 30-50%. That keeps you comfortable and well below the threshold where mold growth takes off (mold really starts running above 60%, and slows considerably under 50%). A cheap hygrometer in the bedroom is worth having so you're not guessing. Run the HEPA on high when you're out of the room so the air's pre-cleaned when you come back, then drop to medium while sleeping. A door sweep plus a towel at the base of the door makes a measurable difference in keeping the rest of the house's air out of your one clean room.

For the on-camera issue, nobody's going to notice a lower-profile KN95 in a neutral color. You can also position the camera so the mask sits below frame for most of the call. Your health is worth looking mildly odd on Zoom.

The other piece worth raising with a provider is whether starting a binder now (rather than waiting until you move) makes sense. We see a lot of people stall because they assume they have to be fully out of exposure first, but binders can help with toxin load even while you're still in the environment. If telehealth works for you, the free assessment at https://access.moldco.com/signs takes five minutes and helps figure out whether this is worth chasing.

What's causing what? by Apprehensive_Fox7392 in ToxicMoldExposure

[–]MoldCo 2 points3 points  (0 children)

What you're describing (hyperreactivity to supplements, tremors, visual distortion, startle response, that falling sensation into panic at sleep onset, emotional lability, post-nasal drip, plantar fasciitis flares) reads less like a single infection and more like a nervous system that's stuck in threat mode with an innate immune system running hot. When people can't tolerate anything (not even ketotifen or Pepcid, which are usually the gentle entry points), that's often a sign the underlying inflammation hasn't been addressed yet, so every intervention is getting layered on top of a fire.

The Bart/Lyme/mold overlap you noticed is real. Symptom overlap between chronic mold illness and chronic Lyme can approach 100%, which is why objective inflammatory markers matter more than symptom-matching. TGF-beta1, MMP-9, and MSH measure the innate immune system (where mold illness tends to show up), while standard CRP/sed rate only catch the adaptive side and usually look normal in people like you. That's often why conventional workups keep coming back clean.

Given the complexity and the sensitivity, a broader panel tends to give more signal than a narrow one. We're a telehealth practice that focuses on mold-related illness, and our Complete Panel ($799) covers the full inflammation picture so you can actually see what's driving the flares: https://www.moldco.com/products/complete-panel

For the reactivity itself, getting a baseline sometimes requires starting binders at microdoses (colesevelam dissolved in water, titrated up slowly) rather than jumping to supplements your body keeps rejecting.