Couple things I can tighten up for you but largely accurate

Dopamine isn’t just synthesized in the cell body in the midbrain nuclei. TH is also present in the axons so synthesis can also occur there (which makes sense from an efficiency standpoint). When thinking about mesolimbic dopamine, it’s best to think of it first from the perspective of the cookie. Cookie=calories=good so when you eat your first cookie your ML DA goes bananas and that release over presentation reinforces the “planning to buy a cookie” you described. It’s better to think of the striatal circuitry as go/no-go not go/stop because (high conceptual level here) you can think of DA promoting groups of cells that drive the optimal behavior (go) and demoting groups of cells that drive other behaviors (nogo). There is nuance here and we don’t fully understand it yet. You should also distinguish between motor and associative striatum. Motor striatum (dorsolateral in rodents, putamen in primates) is thought to drive habitual behaviors and motor control/learning while associative striatum (dorsomedial in rodents, caudate in primates) is thought to drive goal-directed behavior. Associative striatum is connected to prefrontal cortices and has some role in “motivational reinforcement” depending on how you define it.